Background And Objectives: : Occupational noise exposure is a significant risk factor for hearing loss, affecting approximately 5% of the global population. Although noise-induced hearing loss is commonly associated with reduced sensitivity to pure tones, there is limited information regarding when this hearing loss begins after noise exposure. This study aimed to investigate the time of onset of hearing loss in bus drivers exposed to noise for varying durations.
Subjects And Methods: : The study involved 102 bus drivers aged 25-40 years who had been exposed to noise for periods ranging from 6 months to over 10 years. A control group comprising 102 age-matched individuals without noise exposure was also included. Pure-tone audiometry was performed to assess hearing loss, and time of onset of hearing loss relative to noise exposure duration was evaluated.
Results: : Bus drivers experienced only "slight" hearing loss, even after 10 years of noise exposure. By contrast, reduced sensitivity to pure tones was observed after 25 to 48 months of exposure.
Conclusions: : This study confirms that prolonged noise exposure leads to "slight" hearing loss, which can appear as early as 25-48 months after exposure to noise. Among individuals under 40 years of age, significant hearing loss appeared to occur only after 10 or more years of exposure. These findings suggest that the impact of noise on hearing acuity may have been overestimated in previous studies.
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http://dx.doi.org/10.7874/jao.2024.00626 | DOI Listing |
Pediatr Infect Dis J
March 2025
Department of Paediatrics and Child Health, Stellenbosch University and Tygerberg Hospital, Cape Town, South Africa.
Background: Children living with HIV are at higher risk for hearing loss compared to children with HIV-unexposed, uninfected (HUU). There is little known regarding the effects of children living with perinatally-acquired HIV (PHIV) and those living with perinatal HIV exposure but uninfected (PHEU) on central auditory function.
Methods: Children aged 11-14 years who were participating in the Auditory Research in Children with HIV study.
Pediatr Infect Dis J
March 2025
Department of Pediatrics, University of Alabama at Birmingham, Birmingham, Alabama.
Background: Congenital cytomegalovirus is the leading cause of nongenetic sensorineural hearing loss. Treatment with (val)ganciclovir improves audiologic outcomes. Neutropenia is a common adverse event, but correlates that predict who will develop neutropenia have not been identified.
View Article and Find Full Text PDFEnviron Sci Pollut Res Int
March 2025
Department of Environmental Medicine, Faculty of Public Health, Institute of Health Protection, Slovak Medical University in Bratislava, Limbová 2651/12, 833 03, Bratislava, Slovakia.
Approximately 5 years ago, we proposed the establishment of a new category of ototoxicants: environmental ototoxicants, in addition to the recognized categories of occupational and drug-related ototoxicants. Since the publication of our review, the scientific literature has confirmed the potential for hearing impairment (HI) caused by the general population's exposure to various chemicals. However, the extent of this exposure's contribution to the global incidence of hearing loss (HL) has yet to be estimated.
View Article and Find Full Text PDFPsychol Res
March 2025
School of Education, Central China Normal University, Wuhan, China.
This paper explores the cognitive mechanisms of prospective memory in children with hearing impairment through two studies. Study 1, based on questionnaire results, indicates that children with hearing impairment score higher on prospective memory tasks compared to typically developing children. Study 2, derived from experimental outcomes, reveals that children with hearing impairment perform worse on both event-based and time-based prospective memory tasks than their typical hearing peers, with time-based prospective memory showing a more pronounced deficit.
View Article and Find Full Text PDFElife
March 2025
Department of Neuroscience, Georgetown University Medical Center, Washington DC, United States.
Research on brain plasticity, particularly in the context of deafness, consistently emphasizes the reorganization of the auditory cortex. But to what extent do all individuals with deafness show the same level of reorganization? To address this question, we examined the individual differences in functional connectivity (FC) from the deprived auditory cortex. Our findings demonstrate remarkable differentiation between individuals deriving from the absence of shared auditory experiences, resulting in heightened FC variability among deaf individuals, compared to more consistent FC in the hearing group.
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