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Relationship between epicardial adipose tissue thickness and sedentary time, physical activity level, and physical performance in patients with hypertension.
J Hum Hypertens
March 2025
Faculty of Medicine, Department of Cardiology, Karamanoğlu Mehmetbey University, Karaman Training and Research Hospital, Karaman, Turkey.
Epicardial adipose tissue is associated with the development of cardiovascular disease and its increase is positively correlated with blood pressure elevation in hypertensive individuals. In the literature, being physically active has been shown to be effective in the treatment of hypertension and reduction of epicardial adipose tissue thickness. The aim of this study was to evaluate the relationship between epicardial adipose tissue thickness and sedentary time, physical activity level and physical performance in patients with hypertension.
View Article and Find Full Text PDFEpicardial adipose tissue and malignant ventricular arrhythmias in phospholamban p.(Arg14del) variant carriers.
Eur Heart J
March 2025
Department of Cardiology, University Medical Centre Groningen, University of Groningen, Groningen, The Netherlands.
Subepicardial adipose genes contribute to the deterioration of heart failure preserved ejection fraction.
Front Cardiovasc Med
February 2025
Tianjin Key Laboratory of Cardiovascular Emergency and Critical Care, Tianjin Municipal Science and Technology Bureau, Tianjin, China.
Background: The mortality of patients with acute myocardial infarction (MI) raised rapidly in last decade and obesity are becoming the major cause to CAD progression, thus inducing heart failure preserved ejection fraction (HFpEF). However, why visceral adipocytes show different effects on healthy and ageing cardiomyocytes is less known.
Methods: GSE251971 was downloaded and Venn diagram between visceral adipocyte genes genes and DEGs was performed to obtain visceral adipocyte-associated DEGs in heart failure.
NSTEMI Risk Prediction with the Combination of the Biomarkers Epicardial Adipose Tissue and Soluble Suppression of Tumorigenicity 2.
Med Sci Monit
March 2025
Department of Cardiology, Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
BACKGROUND Epicardial adipose tissue (EAT) and soluble suppression of tumorigenicity 2 (sST2) are valuable markers of myocardial fibrosis, but the relationship between EAT and sST2 remains controversial. This study aimed to evaluate the role of combined EAT measurements and levels of sST2 and the risk of major adverse cardiovascular events (MACEs) in patients with diagnosis of non-ST-elevation myocardial infarction (NSTEMI). MATERIAL AND METHODS This was a single-center retrospective observational study.
View Article and Find Full Text PDFCoronary Inflammation and Cardiovascular Events in Patients Without Obstructive Coronary Artery Disease.
Curr Cardiol Rep
March 2025
Paul L. Foster School of Medicine, Texas Tech University Health Sciences Center El Paso, El Paso, TX, USA.
Purpose Of Review: This review evaluates the role of vascular inflammation in patients who develop myocardial infarction with non-obstructive coronary arteries (MINOCA). It also introduces pericoronary adipose tissue (PCAT) and epicardial adipose tissue (EAT) as possible biomarkers for risk prediction in patients with non-obstructive coronary artery disease (CAD).
Recent Findings: PCAT and EAT contribute to the development and progression of coronary artery inflammation and plaque vulnerability.
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