Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1242/bio.048793retraction | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Microglia-mediated neuroinflammation is a key contributor to Parkinson's disease (PD) pathogenesis. Leucine-rich repeat kinase 2 (LRRK2), the leading genetic contributor to both familial and sporadic PD, has been implicated in driving this connection. However, its precise role remains incompletely understood due to technical challenges.
View Article and Find Full Text PDFRetraction: FKBP14 overexpression contributes to osteosarcoma carcinogenesis and indicates poor survival outcome.
Oncotarget
February 2025
Zhejiang Chinese Medical University, Hangzhou 310053, China.
Retraction Note: The oncogenic Golgi phosphoprotein 3 like overexpression is associated with cisplatin resistance in ovarian carcinoma and activating the NF-κB signaling pathway.
J Exp Clin Cancer Res
February 2025
Department of Obstetrics and Gynecology, The First Affiliated Hospital, Sun Yat-Sen University, Zhongshan Second Road 58, Guangzhou, 510700, People's Republic of China.
Regulating Integrin β1 to Restore Gonadotropin-Releasing Hormone-Tanycyte Unit Function in Polycystic Ovary Syndrome-Related Hypothalamic Dysregulation.
Research (Wash D C)
February 2025
Department of Integrative Medicine and Neurobiology, School of Basic Medical Sciences, State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, Institutes of Brain Science, Fudan University, Shanghai 200032, China.
Excessive gonadotropin-releasing hormone (GnRH) is considered to be an initiating factor in the etiology of polycystic ovary syndrome (PCOS). GnRH neuronal axons terminate at the hypothalamic arcuate nucleus and median eminence, where tanycytes, specialized glial cells, have been proposed to modulate GnRH secretion through plasticity. However, the precise role of the "GnRH-tanycyte unit" during the pathological state of PCOS has not been thoroughly explored.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!