Occupational reaching tasks performed with faulty postures may contribute to inefficient movement patterns that could lead to injury. Understanding relationships between posture and muscle activation during reaching tasks may elucidate movement patterns that increase occupational injury risk in workers. This study assessed whether postural factors and muscle activation predict forward reaching movement performance and accuracy. . Predictor variables of forward shoulder posture (FSP), pectoral length, upper (UT), middle (MT) and lower trapezius (LT) and pectoralis major (PM) muscle activation, and UT:PM, MT:PM, and LT:PM co-activation during forward reaching were analysed for 56 individuals. Sequential linear regression equations assessed reaching variance. . For females, FSP, UT activation, and UT:PM co-activation explained 36% of reaction time (RT) variance, and MT:PM co-activation explained 14% of endpoint accuracy variance. For males, MT:PM co-activation explained 17% of movement time (MvT) variance, and FSP, MT:PM co-activation and MT explained 23% of accuracy variance. Increased co-activation was a predictor of movement performance; however, performance outcome variables differed between males (MvT) and females (RT). Muscle co-activation coupled with FSP and posterior shoulder muscle activation resulted in differences in predicting reaching performance variance. Practitioners might consider evaluating these muscle activation and postural factors in occupational reaching tasks.Trial registration: ClinicalTrials.gov identifier: NCT04944745.
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http://dx.doi.org/10.1080/10803548.2025.2466919 | DOI Listing |
JMIR Res Protoc
March 2025
Paseo de los Encomendadores, Faculty of Health Sciences, University of Burgos, Burgos, Spain.
Background: Breast cancer is the second most common cancer in women worldwide. Treatments for this disease often result in side effects such as pain, fatigue, loss of muscle mass, and reduced quality of life. Physical exercise has been shown to effectively mitigate these side effects and improve the quality of life in patients with breast cancer.
View Article and Find Full Text PDFJ Osteopath Med
March 2025
Wood College of Osteopathic Medicine, Marian University, Indianapolis, IN, USA.
Context: Sarcopenia is a disease characterized by low muscle mass and function that places individuals at greater risk of disability, loss of independence, and death. Current therapies include addressing underlying performance issues, resistance training, and/or nutritional strategies. However, these approaches have significant limitations, and chronic inflammation associated with sarcopenia may blunt the anabolic response to exercise and nutrition.
View Article and Find Full Text PDFLiver fibrosis is a global health problem. IL-17A has proven profibrogenic properties in liver disease making it an interesting therapeutic target. IL-17A is regulated by RORγt and produced by Th17 CD4+ and γδ-T cells.
View Article and Find Full Text PDFSci Robot
March 2025
NeuroX Institute and Brain Mind Institute, School of Life Sciences, Swiss Federal Institute of Technology (EPFL), Lausanne, Switzerland.
Rehabilitation robotics aims to promote activity-dependent reorganization of the nervous system. However, people with paralysis cannot generate sufficient activity during robot-assisted rehabilitation and, consequently, do not benefit from these therapies. Here, we developed an implantable spinal cord neuroprosthesis operating in a closed loop to promote robust activity during walking and cycling assisted by robotic devices.
View Article and Find Full Text PDFProc Natl Acad Sci U S A
March 2025
Randall Centre for Cell and Molecular Biophysics and British Heart Foundation Centre of Research Excellence, New Hunt's House, Guy's Campus, King's College London, London SE1 1UL, United Kingdom.
Muscle contraction is driven by myosin motors from the thick filaments pulling on the actin-containing thin filaments of the sarcomere, and it is regulated by structural changes in both filaments. Thin filaments are activated by an increase in intracellular calcium concentration [Ca] and by myosin binding to actin. Thick filaments are activated by direct sensing of the filament load.
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