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| http://dx.doi.org/10.1097/GOX.0000000000006606 | DOI Listing |
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Department of Pharmacology, Arizona Health Sciences Center, University of Arizona, Tucson, AZ, USA.
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View Article and Find Full Text PDFThe CD64/CD28/CD3ζ chimeric receptor reprograms T-cell metabolism and promotes T-cell persistence and immune functions while triggering antibody-independent and antibody-dependent cytotoxicity.
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