Radiation from wireless communication devices inside intelligent connected vehicles has been an expeditious growth of concern regarding possible adverse effects on human health. Due to the significant differences in the working scenarios compared to traditional mobile products, the traditional measuring systems of specific absorption rate (SAR) are not applicable to in-vehicle scenarios. This paper has developed a SAR measurement system and a SAR measurement method, which are suitable for in-vehicle scenarios. Since the measurement hardware and methods are significantly different from traditional systems, it is necessary to assess the measurement uncertainty for the new measurement system. Due to the significant influence of tissue fluid on the SAR, this paper focuses on analyzing the relationship between tissue fluid and SAR. Based on the validated electromagnetic simulation model, linear and quadratic fitting models reflecting the relationship between tissue fluid properties and SAR are established. Then, the uncertainty propagation was realized using both the Guide to the Expression of Uncertainty in Measurement and MCM (Monte Carlo Method) through these models. The results of uncertainty analysis were analyzed in combination with the fitting error. The results of the analyses show that the fitting error of the quadratic measurement model is smaller because there is no simple linear relationship between the tissue fluid properties and the SAR values, and thus, it is more reasonable to use the MCM method to evaluate the uncertainty.
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http://dx.doi.org/10.1063/5.0233227 | DOI Listing |
Gynecol Oncol
January 2025
Departments of Internal Medicine and Immunology, Roswell Park Comprehensive Cancer Center, Buffalo, NY, United States of America; Department of Medicine, Jacobs School of Medicine and Biomedical Sciences, University at Buffalo, Buffalo, NY, United States of America.
Purpose: We observed that the tumor microenvironment (TME) in metastatic epithelial ovarian cancer (EOC) and in other solid tumors can reprogram normal neutrophils to acquire a complement-dependent suppressor phenotype characterized by inhibition of stimulated T cell activation. This study aims to evaluate whether serum markers of neutrophil activation and complement at diagnosis of EOC would be associated with clinical outcomes.
Experimental Design: We conducted a two-center prospective study of patients with newly diagnosed EOC (N = 188).
J Neuroendocrinol
January 2025
Department of Psychology, Columbia University, New York, New York, USA.
Among contributors to diffusible signaling are portal systems which join two capillary beds through connecting veins. Portal systems allow diffusible signals to be transported in high concentrations directly from one capillary bed to the other without dilution in the systemic circulation. Two portal systems have been identified in the brain.
View Article and Find Full Text PDFCureus
December 2024
Otolaryngology, Imperial College London Healthcare National Health Service (NHS) Trust, London, GBR.
We report a case of a 45-year-old gentleman who presented to our major trauma centre after sustaining a penetrating high-pressure paint injection injury to the neck. This rare mechanism of injury is most commonly reported to affect the non-dominant hand, occurring due to the malfunction or misuse of industrial paint machines, causing a piercing soft tissue injury with high-pressure fluid. The unique challenges faced in managing penetrating injuries to the neck are due to the density of vital visceral structures in the region, including major blood vessels and the upper aerodigestive tract.
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View Article and Find Full Text PDFBackground: Juxtaglomerular (JG) cells are sensors that control blood pressure and fluid-electrolyte homeostasis. In response to a decrease in perfusion pressure or changes in the composition and/or volume of the extracellular fluid, JG cells release renin, which initiates an enzymatic cascade that culminates in the production of angiotensin II (Ang II), a potent vasoconstrictor that restores blood pressure and fluid homeostasis. In turn, Ang II exerts a negative feedback on renin release, thus preventing excess circulating renin and the development of hypertension.
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