Communicating artery between posterior circumflex humeral artery and deep brachial artery: case report, literature review and clinical implications. | LitMetric
Purpose: Knowledge of a large communication between posterior circumflex humeral and deep brachial arteries, although infrequent, it is important to avoid damage during a Leechavengvongs procedure or used as recipient artery in free flaps for upper limb reconstruction.
Methods: A dissection of a latex-injected cadaver revealed the presence of a large communication between the posterior circumflex humeral and deep brachial arteries. Furthermore, this communicating artery was observed during a Leechavengvongs procedure. A comprehensive review of the existing literature on the anatomical variations of the posterior circumflex humeral and deep brachial arteries was conducted.
Results: A communicating artery between the posterior circumflex humeral and deep brachial arteries was identified during a routine dissection. This communicating artery establishes a posterior collateral pathway of substantial caliber between the axillary and the brachial arteries. Given its oblique trajectory posteriorly along the humeral shaft, this artery is susceptible to injury during a posterior approach to the humerus. Despite the existence of numerous anatomical variations involving the posterior circumflex humeral and deep brachial arteries, this communicating artery has not been previously described in the literature. Additionally, we report this anatomical variation crossing the surgical field during a Leechavengvongs procedure.
Conclusion: Our findings suggest that a large communicating artery between the posterior circumflex humeral artery and the deep brachial artery has the potential to cross the surgical field and become a source of risk during a posterior approach to the arm. It can also be used as recipient artery in free flaps for upper limb reconstruction.
Background: Acute mitral regurgitation due to papillary muscle rupture is a severe complication of acute myocardial infarction. Transcatheter edge-to-edge repair is emerging as an effective alternative to surgical treatment, with encouraging outcomes. Leaflet adverse events are rare and are associated with relapse of significant mitral regurgitation.
Purpose: Knowledge of a large communication between posterior circumflex humeral and deep brachial arteries, although infrequent, it is important to avoid damage during a Leechavengvongs procedure or used as recipient artery in free flaps for upper limb reconstruction.
Methods: A dissection of a latex-injected cadaver revealed the presence of a large communication between the posterior circumflex humeral and deep brachial arteries. Furthermore, this communicating artery was observed during a Leechavengvongs procedure.
Spontaneous coronary artery dissection (SCAD) is an uncommon but important cause of acute coronary syndrome (ACS), particularly in postpartum women without traditional cardiac risk factors. Our case involves a 29-year-old postpartum woman who presented with severe substernal chest pain eight days after an emergency cesarean section for pregnancy-associated hypertension. Electrocardiography showed ST elevation in the inferior and posterior leads, and coronary angiography revealed a spontaneous dissection in the left circumflex artery (LCx) with an intramural hematoma, alongside a dissection of the right coronary artery (RCA) extending from the ostium to the mid-vessel.
Introduction: Thalamic hemorrhage caused by cerebrovascular disease is unusual. We describe a rare case of thalamic hemorrhage due to rupture of an aneurysm at the feeder of a tentorial dural arteriovenous fistula in the Galen region mimicking a hypertensive hemorrhage.
Case Presentation: A 78-year-old woman with sudden-onset right hemiparesis was diagnosed with left thalamic hemorrhage.
Spontaneous coronary artery dissection (SCAD) is an uncommon non-atherosclerotic etiology of acute coronary syndrome (ACS) characterized by the formation of a false lumen inside the arterial wall, resulting in sudden occlusion of blood flow without any trauma or intervention. The pathogenesis of SCAD is not completely understood, and the association between coronary artery anomalies and SCAD remains unclear. This case study reports a unique occurrence of non-ST-elevation myocardial infarction (NSTEMI) in a 43-year-old female patient.
