Terahertz communication technology based on on-off keying (OOK) direct modulation is vital for sixth-generation communication systems, especially in short-distance and high-rate applications. However, low-order OOK modulation often leads to suboptimal anti-interference capabilities and a heightened demodulation threshold. Here, we propose a high-order direct modulation terahertz communication framework using a wideband time-coding metachip modulator. The modulator leverages the electromagnetic resonance properties within the metaunit structure, with control enabled by gallium arsenide Schottky diodes. By manipulating the timing of voltage pulses applied to these diodes, the equivalent electromagnetic resonance distributions can be precisely regulated in the time domain. This enables independent and accurate control over the amplitude and phase of terahertz harmonics. Leveraging this technique, three high-order modulation schemes-quadrature phase-shift keying, 16-phase-shift keying, and 16-quadrature amplitude modulation- are achieved in a direct modulation and direct detection system, demonstrating the real-time image transmission. The proposed method offers an important way to develop integrated and low-complexity terahertz wireless communication systems.
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http://dx.doi.org/10.1126/sciadv.adq8693 | DOI Listing |
Int J Biochem Cell Biol
December 2024
Institute of Biology and Medicine, College of Life Science and Health, Wuhan University of Science and Technology, Wuhan, Hubei 430081, China; College of Biomedicine and Health, College of Life Science and Technology, Huazhong Agricultural University, Wuhan, Hubei 430070, China. Electronic address:
Considering the high degree of malignancy, recurrence rate and poor prognosis, exploring promising targets is an imperious strategy for colorectal carcinoma therapy. Recent studies have indicated that GABPα plays a role in cancer aggressiveness, but its exact function and regulatory mechanisms in colorectal cancer progression remain unclear. This study aims to explore the biological role of GABPα and its upstream regulator, miR-378a-5p, in modulating cancer progression.
View Article and Find Full Text PDFCell Regen
December 2024
Department of Neurology, Affiliated Hospital of Jining Medical University, 89 Guhuai Road, Jining, 272029, Shandong Province, China.
Peripheral nerve injury (PNI) usually causes severe motor, sensory and autonomic dysfunction. In addition to direct surgical repair, rehabilitation exercises, and traditional physical stimuli, for example, electrical stimulation, have been applied in promoting the clinical recovery of PNI for a long time but showed low efficiency. Recently, significant progress has been made in new physical modulation to promote peripheral nerve regeneration.
View Article and Find Full Text PDFClin Chim Acta
December 2024
Department of Medicine, University of Adelaide, Australia; Endocrine and Metabolic Unit, Royal Adelaide Hospital, Australia; Endocrine and Diabetes Services, The Queen Elizabeth Hospital, Australia.
Background: Corticosteroid-binding globulin (CBG) modulates tissue cortisol availability via modification of cortisol:CBG binding affinity in response to multiple factors, including neutrophil elastase (NE) cleavage of the reactive centre loop (RCL), converting high affinity CBG (haCBG) to low affinity CBG (laCBG). In vitro, glycosylation of the RCL at Asn347 affects NE cleavage susceptibility. To date, no direct measurement of laCBG, which would verify NE cleavage, has been reported.
View Article and Find Full Text PDFBiochim Biophys Acta Mol Basis Dis
December 2024
National Institute of Science Education and Research, School of Biological Sciences, Bhubaneswar, Odisha 752050, India; Homi Bhabha National Institute, Training School Complex, Anushakti Nagar, Mumbai 400094, India. Electronic address:
Maintaining precise levels of FRG1 is vital. It's over-expression is tied to muscular dystrophy, while reduced levels are linked to tumorigenesis. Despite extensive efforts to characterize FRG1 expression and downstream molecular signaling, a comprehensive understanding of its regulation has remained elusive.
View Article and Find Full Text PDFNeurochem Int
December 2024
Basic School of Medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao, 260071, China. Electronic address:
The central endocannabinoid (eCB) system in brain shows a crucial role in the regulation of feeding behaviors, influencing both metabolic and non-metabolic mechanisms of appetite control, which has been paid much attention. Although there are already many review articles discussing eCB modulation of feeding behaviors, our paper attempts to summarize the recent advancements through synapses, circuits, and network in brain. Our focus is on the dual role of eCB signalling in regulating metabolic energy balance and hedonic reward-related feeding.
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