Estimating the actual impact of eating out on calorie intake poses challenges due to the potential endogeneity of eating out. Controlling for endogeneity is essential to obtain more precise estimates. This study estimates the effect of eating out on calorie intake, specifically focusing on dinner among the three daily meals. The endogenous treatment effect model is employed, utilizing restricted-access data from the 2017 to 2020 Korea National Health and Nutrition Examination Survey. To aid model identification, daily precipitation is used as an instrumental variable influencing the decision to eat dinner out. Eating dinner out results in an additional intake of 388.39 Kcal compared to eating at home, whereas the model that does not control for the endogeneity estimates a 261 Kcal increase. The increase in calorie intake from eating dinner out varies significantly depending on individual characteristics. Eating out leads to a substantial amount of calorie intake. Managing the frequency of eating out at a reasonable level could play a crucial role in preventing obesity.
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http://dx.doi.org/10.1177/02601060241297857 | DOI Listing |
Nutrients
January 2025
Department of Nutrition, Food Sciences and Physiology, Center for Nutrition and Research, University of Navarra, 31008 Pamplona, Spain.
Background And Aim: Telomere length (TL) is a key biomarker of cellular aging, with shorter telomeres associated with age-related diseases. Lifestyle interventions mitigating telomere shortening are essential for preventing such conditions. This study aimed to examine the effects of two weight loss dietary strategies, based on a moderately high-protein (MHP) diet and a low-fat (LF) diet on TL in individuals with overweight or obesity.
View Article and Find Full Text PDFNutrients
January 2025
Department of Pediatric Intensive Care, Faculty of Medicine, Mugla Sitki Kocman University, Mugla 48000, Türkiye.
Background: The inability to ensure adequate nutrition for patients, and failure to provide adequate calorie and protein intake, result in malnutrition, leading to increased morbidity and mortality. The present study assesses the two approaches to enteral nutrition-intermittent and continuous enteral feeding-in critically ill pediatric patients in Türkiye to determine the superiority of one method over the other.
Methods: Included in this multicenter prospective study were patients receiving enteral nutrition via a tube who were followed up over a 3-month period.
Nutrients
January 2025
Division of Nutrition, Food & Dietetics, School of Biosciences, University of Nottingham, Leics LE12 5RD, UK.
With the rise of artificial intelligence (AI) in nutrition and healthcare, AI-driven chatbots are increasingly recognised as potential tools for generating personalised diet plans. This study aimed to evaluate the capabilities of three popular chatbots-Gemini, Microsoft Copilot, and ChatGPT 4.0-in designing weight-loss diet plans across varying caloric levels and genders.
View Article and Find Full Text PDFBiomolecules
January 2025
Department of Nutritional Sciences, Auburn University, Auburn, AL 36849, USA.
Obesity is characterized by the enlargement of adipose tissue due to an increased calorie intake exceeding the body's energy expenditure. Changes in the size of adipose tissue can lead to harmful consequences, with excessive fat accumulation resulting in adipocyte hypertrophy and promoting metabolic dysfunction. These adiposity-associated pathologies can be influenced by dietary components and their potential health benefits.
View Article and Find Full Text PDFAntioxidants (Basel)
January 2025
Institute of Clinical Physiology, National Research Council, Via Moruzzi 1, 56124 Pisa, Italy.
Cellular senescence is a state of permanent cell cycle arrest accompanied by metabolic activity and characteristic phenotypic changes. This process is crucial for developing age-related diseases, where excessive calorie intake accelerates metabolic dysfunction and aging. Overnutrition disturbs key metabolic pathways, including insulin/insulin-like growth factor signaling (IIS), the mammalian target of rapamycin (mTOR), and AMP-activated protein kinase.
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