Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1016/j.nuclcard.2024.102067 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Splenic switch-off to assess for vasodilator non-responsiveness.
J Nucl Cardiol
November 2024
Department of Medicine (Cardiology), Mount Sinai Morningside Hospital, Zena and Michael A. Weiner Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, New York, USA; Department of Population Health Science and Policy, Blavatnik Family Women's Health Research Institute, Icahn School of Medicine at Mount Sinai, New York, USA. Electronic address:
Prediction of cardiac allograft vasculopathy using splenic switch-off on myocardial PET.
J Cardiol
July 2024
Department of Cardiology, Tokyo Women's Medical University, Tokyo, Japan.
Background: Heart transplantation (HTx) is a definitive therapy for refractory heart failure. Cardiac allograft vasculopathy (CAV), characterized by diffuse arteriopathy involving the epicardial coronary arteries and microvasculature, is the major cause of death for patients with HTx. N-ammonia positron emission tomography (NH-PET) can offer diagnostic and prognostic utility for CAV.
View Article and Find Full Text PDFSplenic switch-off in [O]HO-positron emission tomography myocardial perfusion imaging using parametric blood flow images.
J Nucl Cardiol
August 2024
Department of Nuclear Medicine and PET-Centre, Aarhus University Hospital, Denmark; Department of Clinical Medicine, Aarhus University, Denmark.
Background: Evaluation of sufficient adenosine response constitutes a significant challenge in myocardial perfusion imaging (MPI). Splenic switch-off in MPI studies denotes a visually (qualitatively) reduced splenic radiotracer signal during adenosine stress and is considered indicative of sufficient cardiac vasodilation. In this study, we examined semi-quantitative and quantitative approaches to splenic switch-off assessment using [O]HO-PET with either summed activity images or calculated parametric splenic blood flow images.
View Article and Find Full Text PDFHow to assess nonresponsiveness to vasodilator stress.
J Nucl Cardiol
June 2024
Department of Medicine (Cardiology), Mount Sinai Morningside Hospital, Zena and Michael A. Weiner Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, New York, USA; Department of Population Health Science and Policy, Blavatnik Family Women's Health Research Institute, Icahn School of Medicine at Mount Sinai, New York, USA. Electronic address:
Myocardial perfusion imaging (MPI) is a powerful tool for the functional assessment of ischemia in patients with suspected or known coronary artery disease (CAD). Given that the diagnostic accuracy and prognostic value of MPI and post-test management are highly dependent on achieving an adequate stress vasodilatory response, it is critical to identify those who may not have adequately responded to vasodilator pharmacological stress agents such as adenosine, dipyridamole, and regadenoson. Caffeine, a potent inhibitor of the adenosine receptor, is a compound that can affect vasodilatory hemodynamics, result in false negative studies, and potentially alter management in cases of inaccurate test results.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!