Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1021/acs.chemrev.4c00712 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Management of wind-turbine blade waste as high-content concrete addition: Mechanical performance evaluation and life cycle assessment.
J Environ Manage
January 2025
Department of Civil Engineering, Escuela Politécnica Superior, University of Burgos, c/ Villadiego s/n, 09001, Burgos, Spain. Electronic address:
The management of end-of-life wind-turbine blades in the coming years will be necessary, as a clear solution for their recycling is yet to be found due to their complex composition. The suitability of their mechanical recycling is therefore evaluated in this paper, obtaining Raw-Crushed Wind-Turbine Blade (RCWTB) for subsequent incorporation in high amounts of up to 10% vol. in concrete, replacing the aggregates to achieve Fiber-Reinforced Concrete (FRC).
View Article and Find Full Text PDFBasic Science and Pathogenesis.
Alzheimers Dement
December 2024
Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.
Background: Women are disproportionately affected by Alzheimer's disease (AD) and exhibit greater AD neuropathology than men. Women possess two X chromosomes, with one randomly silenced across each cell for dosage compensation. X chromosome inactivation (XCI) is not complete, and XCI-escaping genes provide a promising avenue of discovery for biological pathways driving sex-specific AD risk.
View Article and Find Full Text PDFTAT-1, a phosphatidylserine flippase, affects molting and regulates membrane trafficking in the epidermis of C. elegans.
Genetics
December 2024
Department of Molecular Biology, College of Agriculture, Life Sciences and Natural Resources, University of Wyoming, Laramie, Wyoming 82071.
Membrane trafficking is a conserved process required for import, export, movement, and distribution of proteins and other macromolecules within cells. The Caenorhabditis elegans NIMA-related kinases NEKL-2 (human NEK8/9) and NEKL-3 (human NEK6/7) are conserved regulators of membrane trafficking and are required for the completion of molting. Using a genetic approach we identified reduction-of-function mutations in tat-1 that suppress nekl-associated molting defects.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!