AI Article Synopsis

  • Collaborative inhibition is a phenomenon where collaboration can hinder memory recall, influenced by various social and cognitive factors.
  • A meta-analysis examined how gender, relationships, culture, and memory monitoring affect collaborative inhibition, revealing significant moderating effects from these factors.
  • The study confirms that collaborative inhibition is a consistent effect, meaning it regularly occurs under these conditions, yet the specific mechanisms behind it need more research.

Article Abstract

Collaborative inhibition is a counterintuitive phenomenon. While the specific mechanisms through which social factors influence collaborative inhibition remain unclear, this study aims to shed light on the impact of gender, relationships, and culture in order to better understand the factors shaping collaborative inhibition. A meta-analysis was conducted to analyze subgroups of social factors, including collaborative pairing, gender, membership, and culture, as well as cognitive factors like memory monitoring. Collaborative inhibition was found to be a robust effect ( < 0.01), with moderating effects of pairing on gender ( < 0.01), membership ( < 0.01), culture ( < 0.01), and memory monitoring ( < 0.01). The findings indicate that collaborative inhibition is a consistent phenomenon influenced by both social and cognitive factors. Moreover, the study discovered that memory monitoring can successfully moderate collaborative inhibition, although the underlying mechanism requires further investigation.

Download full-text PDF

Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC11428894PMC
http://dx.doi.org/10.3390/bs14090763DOI Listing

Publication Analysis

Top Keywords

collaborative inhibition
28
memory monitoring
16
gender membership
8
membership culture
8
monitoring collaborative
8
social factors
8
pairing gender
8
cognitive factors
8
inhibition
7
collaborative
7

Similar Publications

Hsp70, Hsp90, and ClpB/Hsp100 are molecular chaperones that help regulate proteostasis. Bacterial and yeast Hsp70s and their cochaperones function synergistically with Hsp90s to reactivate inactive and aggregated proteins by a mechanism that requires a direct interaction between Hsp90 and Hsp70 both in vitro and in vivo. and yeast Hsp70s also collaborate in bichaperone systems with ClpB and Hsp104, respectively, to disaggregate and reactivate aggregated proteins and amyloids such as prions.

View Article and Find Full Text PDF

Purpose: The effective compound combination of Bufei Yishen formula III (ECC-BYF III) has shown protective effects against chronic obstructive pulmonary disease (COPD). However, its effect on mitochondrial dysfunction remains unclear. The current study aimed to investigate the effect of ECC-BYF III on mitochondrial dysfunction in COPD mice and elucidate its potential mechanisms.

View Article and Find Full Text PDF

A novel application perspective of the clinical-used drug verapamil on osteoporosis via targeting .

J Orthop Translat

January 2025

Shanghai Key Laboratory of Orthopedic Implants, Department of Orthopedic Surgery, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200011, People's Republic of China.

Background: RANKL and SCLEROSTIN antibodies have provided a strong effective choice for treating osteoporosis in the past years, which suggested novel molecular target identification and therapeutic strategies development are important for the treatment of osteoporosis. The therapeutic effect of verapamil, a drug previously used for cardiovascular diseases, on diabetes was due to the inhibition of TXNIP expression, which has also been reported as a target in mice osteoporosis. Whether verapamil-inhibited TXNIP expression is related to osteoporosis and how it works on the molecular level is worthy to be explored.

View Article and Find Full Text PDF

Ginsenoside Rd (Rd) is a bioactive compound predominantly found in Panax ginseng C.A. Meyer and Panax notoginseng (Burkill) F.

View Article and Find Full Text PDF

IgA nephropathy (IgAN) is related to the balance of gut microbiota. However, it is unclear whether changes in the gut microbiota can cause IgAN or attenuate its progression. This study employed IgAN and human microbiota-associated (HMA)-IgAN models to investigate the impact of IgAN on gut microbiota alteration and the mechanisms by which gut microbiota might trigger IgAN.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!