AI Article Synopsis
- The study investigates the properties of the nodoventricular pathway (NVP) and its role in supraventricular tachycardia events.
- During the research, a specific type of premature ventricular contraction (PVC) was observed to effectively terminate the tachycardia, revealing unique behavior in the NVP.
- The findings suggest that the behavior of the NVP contributes to the mechanism of slow-fast AV nodal reentrant tachycardia (AVNRT), with the timing of the PVC affecting the conduction properties and ultimately the termination of the tachycardia.
Article Abstract
Introduction: The decremental properties of the nodoventricular pathway (NVP) are uncertain.
Methods And Results: During short RP supraventricular tachycardia, a His-refractory premature ventricular contraction (PVC) consistently terminated the tachycardia without atrial capture immediately after the PVC. Whereas a slightly earlier PVC failed to reset the subsequent His but terminated the tachycardia without atrial capture one cycle later.
Conclusion: These observations are diagnostic of slow-fast atrioventricular nodal reentrant tachycardia (AVNRT) with a bystander concealed-NVP and can be explained by decremental properties in the NVP itself; greater prematurity of the PVC resulted in more decremental conduction over the NVP, causing the AVNRT termination one cycle later.
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| http://dx.doi.org/10.1111/jce.16430 | DOI Listing |
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