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Lateral collateral ligament complex insufficiency in recalcitrant lateral epicondylitis: MRI evaluation with arthroscopic findings. | LitMetric

Lateral collateral ligament complex insufficiency in recalcitrant lateral epicondylitis: MRI evaluation with arthroscopic findings.

Int Orthop

Department of Orthopedic Surgery, College of Medicine, Asan Medical Center, Ulsan University, 88 Olympic-ro 43 gil, Pungnap 2(i)-dong, Songpa-gu, Seoul, 05505, Korea.

Published: November 2024

AI Article Synopsis

  • The study aimed to analyze the relationship between chronic recalcitrant lateral epicondylitis (LE) and accompanying pathologies using MRI and arthroscopic findings.
  • Out of 49 patients, the study classified them into two groups based on lateral collateral ligament (LCL) complex injuries and found significant differences in symptom duration and steroid injection history between the groups.
  • The research concluded that recalcitrant LE is often linked to additional pathologies like LCL insufficiency and elbow plica lesions, with certain arthroscopic findings serving as indicators for these conditions.

Article Abstract

Purpose: To evaluate the concomitant pathology in recalcitrant LE using MRI with arthroscopic finding correlation.

Methods: A total of 49 patients were diagnosed with chronic recalcitrant LE and divided into two: LCL complex-intact and LCL complex-involved groups by evaluating MRI as confirmed by a radiologist. Patient information for the history of steroid injection and symptom duration was extracted from the medical records. Arthroscopic images taken during arthroscopic extensor carpi radialis brevis release were evaluated to assess the quality of lateral capsule and concomitant plica.

Results: A total of 24 and 25 patients were included in the LCL-intact and LCL-involved groups, respectively. Among them, seven had complete RCL tears recorded in the LCL-involved group. Symptom duration (15 ± 9 vs. 22 ± 13, p = 0.029) and the number of steroid injections (3 ± 2 vs. 5 ± 3, p = 0.040) were significantly higher in the LCL-involved group than that in the LCL-intact group. A capsular tear was detected for 5 (20%) patients in the LCL-intact and 14 (56%) in the LCL-involved group (p = 0.027). Concomitant plica was observed in 15 (62%) patients in LCL-intact and seven (28%) in the LCL-involved group (p = 0.015). RC joint widening was observed in four patients in the LCL-involved group.

Conclusion: The recalcitrant LE is highly a concomitant pathology including LCL complex insufficiency and pathologic elbow plica lesion. The risk factors of LCL insufficiency associated with refractory LE may include multiple steroid injections. Arthroscopic finding such as capsular tears and elbow drive-through signs can be suspected signs for LCL complex insufficiency.

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Source
http://dx.doi.org/10.1007/s00264-024-06305-2DOI Listing

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