Levites et al. demonstrate that mouse models of Alzheimer disease (AD), exhibiting amyloid-beta (Αβ) plaque formation, share Αβ responsome proteins with humans. Their work underscores the value of these models in studying Αβ aggregation, cellular vulnerability, and early-stage AD pathology.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC11384129 | PMC |
| http://dx.doi.org/10.1016/j.xcrm.2024.101683 | DOI Listing |
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