The Foix-Alajouanine syndrome was originally reported by these authors in 1926, as rapidly progressive vasculitis on the background of a viral infection. The pathology was represented by the huge, more than 10 times, dilation either of the lumen, or the walls of the spinal vessels, either of the arteries, or the veins. There were no signs of thrombosis, no malformations. Massive necrosis was observed in the spinal cord. Though plenty of observations of the syndrome were reported over the past 100 years, most of them deal with arteriovenous malformations and/or thrombosis, which had not been revealed originally. We present the case of spinal viral vasculitis detected by means of spinal MR-angiography. The undoubted viral etiology of vasculitis allows us to attribute this observation to Foix-Alajouanine syndrome.
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http://dx.doi.org/10.17116/jnevro202412408247 | DOI Listing |
J Formos Med Assoc
December 2024
Department of Orthopedic Oncology, The Third Affiliated Hospital of Southern Medical University, Guangzhou, 510630, Guangdong, PR China. Electronic address:
J Formos Med Assoc
December 2024
Department of Medical Imaging, National Taiwan University Hospital, Taipei, Taiwan. Electronic address:
Kyobu Geka
October 2024
Department of Cardiovascular Surgery, Amagasaki General Medical Center, Amagasaki, Japan.
A 53-year-old male, diagnosed with Stanford A acute aortic dissection, underwent a total arch replacement with a conventional elephant trunk. During the extubation phase, paraplegia and sensory disturbance were identified, prompting suspicion of spinal cord injury (SCI) attributed to thrombosis of the false lumen and subsequent obstruction of the segmental arteries. Apixaban was administered in addition to spinal drainage, meticulous blood pressure management, and administration of mannitol, edaravone, naloxone, and prednisolone.
View Article and Find Full Text PDFRinsho Shinkeigaku
December 2024
Department of Neurology and Stroke Treatment, Japanese Red Cross Kyoto Daiichi Hospital.
We present a case of a 53-year-old man who was admitted with lower back pain and bilateral lower limb weakness. Neurologically, he exhibited paralysis of both lower limbs, complete sensory loss below the 10th thoracic spinal level, and bladder and rectal dysfunction. Spinal MRI revealed intramedullary high-signal lesions extending from the 10th vertebral level to the conus medullaris on diffusion-weighted and T-weighted images.
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