Herpes simplex virus-1 (HSV-1) infection of cultured human neural cells causes the accumulation of a host cell-encoded nuclear protein identified as a 57,000 mol.wt. stress protein by monoclonal antibody TI56. This protein is cell cycle-related in fibroblast, may mediate host cell control during HSV infection and could play a role in the regulation of HSV latency.
Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1016/0304-3940(85)90484-7 | DOI Listing |
Publication Analysis
Top Keywords
cultured human
8
human neural
8
neural cells
8
herpes simplex
8
cells accumulate
4
accumulate heat-shock
4
protein
4
heat-shock protein
4
protein acute
4
acute herpes
4
Similar Publications
Want AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!