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Reply to Editorial Comment on "Letter to the Editor on "Trends in Urology Residency Applications: Results from The American Urological Association Medical Student Survey from 2022 to 2024".
Urology
March 2025
University of Kansas, Department of Urology, Kansas City, KS. Electronic address:
Immo-bile-izing CD8 T cell anti-tumor immunity.
Immunity
March 2025
Blacktown Clinical School, Western Sydney University, Sydney, NSW 2148, Australia; Storr Liver Centre, Westmead Institute for Medical Research, Sydney, NSW 2145, Australia; Blacktown Mt Druitt Hospital, Sydney, NSW 2148, Australia. Electronic address:
Hepatocellular carcinoma is poorly responsive to immune checkpoint blockade. In a recent issue of Science, Varanasi et al. reveal how bile acids dampen anti-tumor CD8 T cell responses in the liver, contributing to cancer progression and poor immunotherapy outcomes.
View Article and Find Full Text PDFRIPK3: The Deadpool of neuronal cell death proteins?
Immunity
March 2025
Laboratory of Neurological Infections and Immunity, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 903 S. 4th Street, Hamilton, MT 59840, USA. Electronic address:
Receptor interaction kinase 3 (RIPK3) is a key mediator of necroptosis, initiating programmed cell death with mercenary efficiency in multiple neurodegenerative diseases. However, in this issue of Immunity, Estevez et al. demonstrate that RIPK3, like the character Deadpool, exists in duality and can also be a force for good by preventing excitotoxic neuronal death during orthoflavivirus encephalitis.
View Article and Find Full Text PDFThe CREscendoing promise of HDAC targeting to limit atherosclerosis.
Immunity
March 2025
Department of Laboratory Medicine, Medical University of Vienna, Vienna, Austria. Electronic address:
The factors that modulate the inflammatory response in atherosclerosis are not well defined. In this issue of Immunity, Asare et al. examine the impact of a cis-regulatory element (CRE) that controls expression of HDAC9 and find that HDAC9-mediated deacetylation of NLRP3 might be the mechanism by which genetic variants in this conserved CRE influence the inflammation associated with human atherosclerosis.
View Article and Find Full Text PDFReply: Opioids Intertwined With Psychology: Hidden Effects in the Treatment of Patients With IBD.
Inflamm Bowel Dis
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Leeds Gastroenterology Institute, St. James's University Hospital, Leeds, UK.
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