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Thoracic aortic mural thrombi (TAMT) are rare but serious conditions that can lead to serious complications, including death, and are often linked to hypercoagulability.
A case study is presented involving an 87-year-old man with TAMT who also had a prothrombin mutation and several health issues that contributed to his hypercoagulable state.
The authors call for more research on how to treat TAMTs effectively and stress the importance of timely testing for hypercoagulable conditions to prevent future health crises.
Article Abstract
Thoracic aortic mural thrombi (TAMT) are rare yet a significant cause of emboli and mortality. Hypercoagulability is thought to play a role in pathogenesis. A common association is prothrombin G20210A mutation. We present a case of an 87-year-old man with an incidentally found TAMT in the setting of prothrombin mutation, metastatic prostate cancer, and a myeloproliferative disorder. The patient had several causes activating Virchow's hypercoagulability principle, contributing to a centrally located clot. Because of its paucity in the literature, we advocate for further research concerning treatment modalities of TAMTs as well as an additional and timely workup for hypercoagulable states to prevent further calamity.
