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http://dx.doi.org/10.4088/PCC.23br03678 | DOI Listing |
Prim Care Companion CNS Disord
May 2024
Department of Psychiatry, Texas Tech University Health Science Center at Odessa/Permian Basin Odessa, Midland, Texas.
Neurocirugia (Astur : Engl Ed)
May 2021
Servicio de Neurología, Hospital Virgen de la Salud, Toledo, España.
Camptocormia is an involuntary flexion between 15-45° of the thoracolumbar spine when standing, walking or sitting, which disappears completely in the supine position. Is associated to Parkinson's disease and other neurodegenerative conditions, being its physiopatology unknown. We report three cases of atypical anti-psychotic induced camptocormia.
View Article and Find Full Text PDFTremor Other Hyperkinet Mov (N Y)
April 2019
Department of Neurology, Post Graduate Institute of Medical Education and Research, Chandigarh, IN.
Background: Camptocormia is defined as forward flexion of the spine that manifests during walking and standing and disappears in recumbent position. The various etiologies include idiopathic Parkinson's disease, multiple system atrophy, myopathies, degenerative joint disease, and drugs.
Case Report: A 67-year-old diabetic female presented with bradykinesia and camptocormia that started 1 year prior to presentation.
Camptocormia is a rare, involuntary movement disorder, presenting as truncal flexion while standing or walking, and is mainly observed as a feature of Parkinson's disease (PD) and primary dystonia. Deep brain stimulation (DBS) of the globus pallidus internus is effective for refractory camptocormia observed with PD or dystonia. However, the effectiveness of pallidotomy for camptocormia has not been investigated.
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