A case of femoral palsy associated with chronic pain sensible to Carbamazepine, secondary to iliacus haematoma during anticoagulant therapy is reported. Late femoral nerve decompression followed by transcutaneous neurostimulation permit the normalization of the sensory nerve conduction and a complete clinical recovery. The localizations of hemorrhage and nervous compression are discussed. The necessity of a nerve decompression when features of nervous conduction persist is emphasized. Clinical symptomatology, Carbamazepine and transcutaneous neurostimulation efficacity, electrophysiological features are correlated with a central deafferentation state caused by an incomplete and heterogeneous lesion of the sensory nerve conduction pathways.
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