AI Article Synopsis
- Acute acalculous cholecystitis (AAC) is a condition where the gallbladder becomes inflamed without the presence of gallstones, and it is extremely rare for it to be caused by the hepatitis A virus (HAV).
- A case study of a 41-year-old patient revealed that she experienced significant abdominal pain and vomiting, and after surgery, it was found that her AAC was due to an acute HAV infection, despite the successful removal of her inflamed gallbladder.
- The discussion highlights the need for conservative management in cases of HAV induced AAC rather than immediate surgery, calling for more cases and research to better understand this rare presentation and its treatment options.
Article Abstract
Introduction: acute acalculous cholecystitis (AAC) is defined as gallbladder inflammation without the presence of stones. Contrary, hepatitis A virus (HAV) can present with different symptoms; however, HAV causing and presenting as AAC is rare.
Case Presentation: 41-year-old previously healthy patient presented with right upper quadrant abdominal pain. The pain was persistent and associated with vomiting and laboratory tests showed elevated bilirubin. Laparoscopic cholecystectomy showed inflamed gallbladder with no stones and intraoperative cholangiography showed no abnormalities. Day one post-operation, while the pain resolved, labs showed elevated liver function tests and hepatitis workup showed acute HAV infection attributing her presentation to HAV induced AAC.
Discussion: AAC is usually caused by stasis of the gallbladder due to different causes; however, HAV induced AAC has been rarely reported. While cholecystectomy is the mainstay treatment for AAC, this might not be the case for HAV induced AAC. For instance, unless there is necrotic gallbladder or persistence of symptoms, AAC can be managed conservatively in this case. Even though our diagnosis was cleared post-operatively, had we knew the diagnosis of HAV induced AAC before, we would have still opt for surgery due to the severity and persistence of pain.
Conclusion: More cases should be reported and more studies should be done to further define the presentation and management of HAV induced AAC.
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Source |
|---|---|
| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC11059507 | PMC |
| http://dx.doi.org/10.1016/j.ijscr.2024.109687 | DOI Listing |
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