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FIRRM and FIGNL1: partners in the regulation of homologous recombination. | LitMetric

FIRRM and FIGNL1: partners in the regulation of homologous recombination.

Trends Genet

Department of Medical Oncology, University Medical Center Groningen, University of Groningen, Hanzeplein 1, 9713GZ, Groningen, The Netherlands. Electronic address:

Published: June 2024

AI Article Synopsis

Article Abstract

DNA repair through homologous recombination (HR) is of vital importance for maintaining genome stability and preventing tumorigenesis. RAD51 is the core component of HR, catalyzing the strand invasion and homology search. Here, we highlight recent findings on FIRRM and FIGNL1 as regulators of the dynamics of RAD51.

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Source
http://dx.doi.org/10.1016/j.tig.2024.02.007DOI Listing

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