Fully understanding the impact of the human retrotransposon L1 requires that each of ∼500,000 L1 copies be evaluated as a potentially unique genomic entity. In this issue of Cell Genomics, Lanciano et al. strive toward this goal, illuminating the reciprocal regulatory influence between individual L1s and their genomic integration sites.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC10879126 | PMC |
| http://dx.doi.org/10.1016/j.xgen.2024.100504 | DOI Listing |
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