Optical injection locking generally occurs when light from a master laser is unidirectionally injected into a slave laser, such that the injected light overcomes spontaneous emission inside the cavity, and forces the slave laser to behave as a frequency copy of the master. Here, we study the limits of stability for optically pre-amplified optical injection locking in the case of large added noise on the input field and in the presence of a phase locked loop which minimizes the frequency offset between master and slave lasers. We present a set of modified rate equations which we use to describe the physics of the system near the limit of stable injection locking, and report on phase slips which occur due to injected noise momentarily destabilizing the system. We then provide experimental evidence to support the behavior seen in simulation, and are able to successfully recover a CW wave at -80 dBm black box input power (-70 dBm for phase slip free operation), providing 20 dBm of output power from the injection locked slave laser.
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http://dx.doi.org/10.1364/OE.509284 | DOI Listing |
An ultra-narrow-linewidth laser is a core device in fields such as optical atomic clocks, quantum communications, and microwave photonic oscillators. This paper reports an ultra-narrow-linewidth self-injection locked semiconductor laser, which is realized through optical feedback from a high-Q (258 million) Fabry-Perot (FP) cavity constructed with three mirrors, generating an output power of 12 mW. Employing a delay self-heterodyne method based on a signal source analyzer, the phase noise of the laser is -129 dBc/Hz at 100 kHz offset frequency, with an intrinsic linewidth of 3 mHz.
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Heart failure with preserved ejection fraction (HFpEF) remains a major public health burden with increasing prevalence but only few effective therapies. Endothelial dysfunction and inflammation are identified as pathophysiological drivers of HFpEF disease progression. MicroRNAs are increasingly recognized as key regulators of these pathological processes, while antimiR-based therapies have been emerged as promising therapeutics in mice and humans.
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