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Repolarization Dispersion and Phase 2 Re-Entry: One Step Closer to Deciphering Brugada Syndrome?
JACC Clin Electrophysiol
July 2024
Cardiac Electrophysiology Section, Division of Cardiovascular Medicine, Hospital of the University of Pennsylvania, Philadelphia, Pennsylvania, USA. Electronic address:
Dislodgeable Foliar Residue Measurements and Assessment of Dermal Exposure to Captan for Workers in Apple Orchards.
Environ Sci Technol
August 2024
Univ. Bordeaux, CNRS, Bordeaux INP, EPOC, UMR 5805, F-33600 Pessac, France.
Captan dislodgeable foliar residues (DFRs) were determined by following the applications of this fungicide in an apple orchard. The study comprised an investigation of the variability of captan DFR values and 14 days of DFR monitoring to assess kinetic modeling. A method combining solid-phase microextraction (SPME) gas chromatography and high-resolution mass spectrometry (GC-QTOF-MS) was developed for the quantification of captan residues from DFR aqueous extracts.
View Article and Find Full Text PDFDemonstration of Arrhythmia Substrate-Associated Dispersion of Repolarization by Epicardial Unipolar Mapping in Brugada Syndrome.
JACC Clin Electrophysiol
July 2024
Division of Arrhythmia, Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.
Article Synopsis
- The study investigates epicardial unipolar mapping in patients with Brugada syndrome (BrS) and compares it to patients with cardiomyopathy to identify unique characteristics.
- Findings reveal that patients with BrS have significantly higher J-wave amplitudes and a greater repolarization time dispersion index, which can be indicative of the syndrome with high sensitivity and specificity.
- The research highlights that the steep repolarization gradients observed in BrS patients may contribute to arrhythmias, suggesting a unique electrical profile that differentiates it from cardiomyopathy.
Phase-2 Re-Entry in Brugada Syndrome: More to Understand Than "Just" I.
JACC Clin Electrophysiol
December 2023
Department of Biomedical Sciences, University of Copenhagen, Copenhagen, Denmark. Electronic address:
Ventricular Tachycardia Due to Triggered Activity: Role of Early and Delayed Afterdepolarizations.
JACC Clin Electrophysiol
February 2024
Department of Medicine, Division of Cardiology and the Greenberg Institute for Cardiac Electrophysiology, Department of Medicine, Cornell University Medical Center, New York, New York, USA.
Most forms of sustained ventricular tachycardia (VT) are caused by re-entry, resulting from altered myocardial conduction and refractoriness secondary to underlying structural heart disease. In contrast, VT caused by triggered activity (TA) is unrelated to an abnormal structural substrate and is often caused by molecular defects affecting ion channel function or regulation of intracellular calcium cycling. This review summarizes the cellular and molecular bases underlying TA and exemplifies their clinical relevance with selective representative scenarios.
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