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http://dx.doi.org/10.1016/j.scib.2023.09.019 | DOI Listing |
Neural Regen Res
July 2025
The National Clinical Research Center for Geriatric Disease, Xuanwu Hospital, Capital Medical University, Beijing, China.
JOURNAL/nrgr/04.03/01300535-202507000-00028/figure1/v/2024-09-09T124005Z/r/image-tiff Alzheimer's disease is characterized by deposition of amyloid-β, which forms extracellular neuritic plaques, and accumulation of hyperphosphorylated tau, which aggregates to form intraneuronal neurofibrillary tangles, in the brain. The NLRP3 inflammasome may play a role in the transition from amyloid-β deposition to tau phosphorylation and aggregation.
View Article and Find Full Text PDFSci Bull (Beijing)
November 2023
The Florey Neuroscience Institute, The University of Melbourne, Melbourne VIC 3052, Australia. Electronic address:
Leg Med (Tokyo)
March 2003
Molecular Neuroscience Research Center, Shiga University of Medical Science, Tsukinowachoo 18-1, Otsu, Shiga 520-2192, Japan.
Puromycin-sensitive alanyl aminopeptidase (PSA, EC 3.4.11.
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