Clostridium innocuum is a Gram-positive anaerobic spore-forming bacillus that has been identified as part of the normal intestinal microbiota. This bacterium has been rarely associated with human infections, and only few severe infections have been reported until now. In this work, we report on four patients with bacteremia due to C. innocuum, which were well identified by MALDI-TOF MS. Moreover, a review of the previous published cases of bacteremia due to this anaerobic bacterium has been performed.

Download full-text PDF

Source
http://dx.doi.org/10.1016/j.anaerobe.2023.102771DOI Listing

Publication Analysis

Top Keywords

clostridium innocuum
8
bacteremia clostridium
4
innocuum analysis
4
analysis cases
4
cases literature
4
literature review
4
review clostridium
4
innocuum gram-positive
4
gram-positive anaerobic
4
anaerobic spore-forming
4

Similar Publications

Identifying the Involvement of Gut Microbiota in Retinal Vein Occlusion by Mendelian Randomization and Genetic Correlation Analysis.

Transl Vis Sci Technol

January 2025

Department of Otolaryngology & Head and Neck Surgery, Wuhan No.1 Hospital, Wuhan, Hubei, China.

Purpose: Previous researches have suggested an important association between gut microbiota (GM) and vascular pathologies such as atherosclerosis. This study aimed to explore the association between 196 GM taxa and retinal vein occlusion (RVO).

Methods: This study used Mendelian randomization (MR), linkage disequilibrium score regression (LDSC), and polygenic overlap analysis.

View Article and Find Full Text PDF

Background: Lipoma, a benign tumor derived from mesenchymal tissue, significantly affects patients' physical and psychological wellbeing. Increasing evidence points to a strong link between the gut microbiome (GM) and lipoma incidence. This study utilizes Mendelian Randomization (MR) to assess the potential causal relationships between the GM and lipoma development.

View Article and Find Full Text PDF

Gut Microbial Utilization of the Alternative Sweetener, D-Allulose, via AlsE.

bioRxiv

November 2024

College of Computer, Mathematical and Natural Sciences, University of Maryland, College Park, Maryland, USA.

D-allulose, a rare sugar with emerging potential as a low-calorie sweetener, has garnered attention as an alternative to other commercially available alternative sweeteners, such as sugar alcohols, which often cause severe gastrointestinal discomfort. D-allulose-6-phosphate 3-epimerase (AlsE) is a prokaryotic enzyme that converts D-allulose-6-phosphate into D-fructose-6-phopshate, enabling its use as a carbon source. However, the taxonomic breadth of AlsE across gut bacteria remains poorly understood, hindering insights into the utilization of D-allulose by microbial communities.

View Article and Find Full Text PDF
Article Synopsis
  • - Levels of progesterone increase after ovulation and are vital for supporting pregnancy, but gut microbiota can influence these levels by affecting the hormone's recycling in the liver and gut.
  • - A specific gut bacterium plays a key role in metabolizing progesterone into a neurosteroid, which does not have progestogenic activity; researchers identified the enzyme responsible for this conversion.
  • - In experiments with female mice, certain antibiotics helped restore progesterone levels, and prolonged antibiotic use disrupted ovarian follicle development, suggesting a link between gut bacteria and progesterone resistance in women.
View Article and Find Full Text PDF

Previous studies have suggested a link between the gut microbiome and hypertension-related traits like blood pressure. However, these reports are often limited by weak causal evidence. This study investigates the potential causal association between gut microbiota and hypertension-related traits using Mendelian randomization with summary data from genome-wide association studies.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!