Askola fortuita sp. nov. is described based on male imagos from Southeastern Brazil, near Caparaó National Park. The new species is somewhat similar to A. froehlichi Peters, 1969 in regards to development of the male compound eyes and relative length of penis, but can be readily distinguished from this and other species of the genus by the following characteristics: dorsal portion of compound eyes widely meeting on meson of head; forewings hyaline; femora white, with a submedial mark and an apical band, both orangish brown; abdominal coloration mostly translucent white; genitalia with styliger plate washed with brown and red laterally, forceps segment I reddish-brown on basal ¼ , remainder translucent white, penes yellowish brown; penes not extending beyond posterior margin of styliger plate.
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http://dx.doi.org/10.11646/zootaxa.5315.2.8 | DOI Listing |
Replacement names are proposed for the following six genera: Ahrenserica Allsopp & Schoolmeesters, replacement name for Orthoserica Brenske, 1900 (Sericini); Brenskecnemus Allsopp & Schoolmeesters, replacement name for Holocnemus Brenske, 1894 (Melolonthini); Dombrocnemis Allsopp & Schoolmeesters, replacement name for Toxocnemis Dombrow, 2002 (Hopliini); Lazomba Allsopp & Schoolmeesters, replacement name for Zomba Lacroix, 2006 (Pachydemini); Perabula Allsopp & Schoolmeesters, replacement name for Rabula Péringuey, 1902 (Hopliini); Phillsoppia Allsopp & Schoolmeesters, replacement name for Leptocnemis Dombrow, 2001 (Hopliini). Rabula morosa Péringuey, 1902 is designated as the type species of Perabula. Miotemna Lacordaire, 1856 is an unnecessary replacement name for Octotemna Blanchard, 1850 (Sericini).
View Article and Find Full Text PDFPLoS Pathog
November 2024
Department of Molecular Microbiology, Washington University School of Medicine in St. Louis; St. Louis, Missouri, United States of America.
Acinetobacter baumannii causes life-threatening infections that are becoming difficult to treat due to increasing rates of multi-drug resistance (MDR) among clinical isolates. This has led the World Health Organization and the CDC to categorize MDR A. baumannii as a top priority for the research and development of new antibiotics.
View Article and Find Full Text PDFNat Commun
April 2024
Division of Epidemiology, Department of Medicine, Vanderbilt-Ingram Cancer Center, Vanderbilt Epidemiology Center, Vanderbilt University Medical Center, Nashville, TN, USA.
Genome-wide association studies (GWAS) have identified more than 200 common genetic variants independently associated with colorectal cancer (CRC) risk, but the causal variants and target genes are mostly unknown. We sought to fine-map all known CRC risk loci using GWAS data from 100,204 cases and 154,587 controls of East Asian and European ancestry. Our stepwise conditional analyses revealed 238 independent association signals of CRC risk, each with a set of credible causal variants (CCVs), of which 28 signals had a single CCV.
View Article and Find Full Text PDFEpilepsia Open
June 2024
Chalfont Centre for Epilepsy, Chalfont St Peter, Bucks, UK.
Objective: To assess asymptomatic rates and severity of SARS-CoV-2 infection in people with epilepsy and their healthcare workers in a long-term care facility which had implemented weekly surveillance testing between April 2020 and June 2022.
Methods: Questionnaires focused on objective and subjective COVID-19 symptoms for people with epilepsy residing in and their healthcare workers at the Chalfont Centre for Epilepsy in June 2022. Demographic information, comorbidities, and seizure frequency were gathered from medical records.
Cell Death Dis
March 2024
Division of Cancer Research, Peter MacCallum Cancer Centre, Melbourne, VIC, 3000, Australia.
Caspase-2, one of the most evolutionarily conserved members of the caspase family, is an important regulator of the cellular response to oxidative stress. Given that ferroptosis is suppressed by antioxidant defense pathways, such as that involving selenoenzyme glutathione peroxidase 4 (GPX4), we hypothesized that caspase-2 may play a role in regulating ferroptosis. This study provides the first demonstration of an important and unprecedented function of caspase-2 in protecting cancer cells from undergoing ferroptotic cell death.
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