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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC10382119 | PMC |
| http://dx.doi.org/10.1161/JAHA.123.030495 | DOI Listing |
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Introduction: Type 1 diabetes is a chronic autoimmune condition characterized by the selective destruction of insulin-producing beta cells in the pancreas. The etiology of T1D is multifactorial, with a combination of genetic susceptibility and environmental triggers believed to underlie beta-cell destruction. Preserving and prolonging beta-cell function in T1D is a pivotal therapeutic objective that can mitigate disease progression and improve glycemic control.
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