AI Article Synopsis

  • The centrosome is crucial for cell organization, stability, and the formation of cilia, and recent findings indicate it might also be involved in local protein synthesis.
  • Researchers discovered that TDP-43, an RNA-binding protein linked to neurodegenerative diseases, is localized at the centrosome throughout the cell cycle, which was confirmed through advanced imaging techniques.
  • The study identifies TDP-43's interaction with centrosomal mRNAs and proteins, suggesting that its dysfunction in the centrosome may play a role in the neurodegenerative processes associated with TDP-43-related diseases.

Article Abstract

The centrosome, as the main microtubule organizing centre, plays key roles in cell polarity, genome stability and ciliogenesis. The recent identification of ribosomes, RNA-binding proteins and transcripts at the centrosome suggests local protein synthesis. In this context, we hypothesized that TDP-43, a highly conserved RNA binding protein involved in the pathophysiology of amyotrophic lateral sclerosis and frontotemporal lobar degeneration, could be enriched at this organelle. Using dedicated high magnification sub-diffraction microscopy on human cells, we discovered a novel localization of TDP-43 at the centrosome during all phases of the cell cycle. These results were confirmed on purified centrosomes by western blot and immunofluorescence microscopy. In addition, the co-localization of TDP-43 and pericentrin suggested a pericentriolar enrichment of the protein, leading us to hypothesize that TDP-43 might interact with local mRNAs and proteins. Supporting this hypothesis, we found four conserved centrosomal mRNAs and 16 centrosomal proteins identified as direct TDP-43 interactors. More strikingly, all the 16 proteins are implicated in the pathophysiology of TDP-43 proteinopathies, suggesting that TDP-43 dysfunction in this organelle contributes to neurodegeneration. This first description of TDP-43 centrosomal enrichment paves the way for a more comprehensive understanding of TDP-43 physiology and pathology.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC10473568PMC
http://dx.doi.org/10.1093/brain/awad228DOI Listing

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