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Wet-Cupping's Impact on Pancreatitis Induced by Hypertriglyceridemia: A case study and brief literature review.
Sultan Qaboos Univ Med J
November 2024
Internal Medicine Residency Training Program, Oman Medical Specialty Board, Muscat, Oman.
Article Synopsis
- Familial hypertriglyceridemia is a genetic disorder that causes high levels of triglycerides in the blood, which can lead to acute and chronic pancreatitis.
- A 34-year-old male patient in Muscat, Oman, diagnosed with this condition experienced a significant reduction in triglycerides and a decrease in hospital admissions for pancreatitis after starting cupping therapy.
- This case report suggests that cupping therapy may help manage familial hypertriglyceridemia and lower the risk of pancreatitis.
Short-term clinical and biochemical responses following treatment with dapagliflozin or ertugliflozin in horses with hyperinsulinemia: A retrospective case series.
Domest Anim Endocrinol
January 2025
School of Veterinary Medicine, Murdoch University, Murdoch, Western Australia, Australia.
The metabolic and lipid profiles of horses treated with sodium-glucose cotransporter 2 inhibitors are not well understood. This retrospective study evaluated blood parameters in hyperinsulinemic horses treated with either ertugliflozin (0.05 mg/kg) or dapagliflozin (0.
View Article and Find Full Text PDFResolution of Lipemia Retinalis With Lifestyle Modification.
J Vitreoretin Dis
August 2024
Department of Ophthalmology & Visual Neurosciences, Minneapolis, MN, USA.
To describe a case of lipemia retinalis, a rare ocular manifestation of hypertriglyceridemia that is characterized by a creamy-white to salmon-colored appearance to the fundus. A 55-year-old man was referred for an evaluation for diabetic retinopathy and was subsequently found to have marked lipemia retinalis. The patient's triglyceride levels were 3141 mg/dL; therefore, treatment was initiated with high-intensity statin therapy and lifestyle modifications.
View Article and Find Full Text PDFFIBRINOGEN-LIKE PROTEIN 2 PROTECTS THE AGGRAVATION OF HYPERTRIGLYCERIDEMIA ON THE SEVERITY OF HYPERTRIGLYCERIDEMIA ACUTE PANCREATITIS BY REGULATING MACROPHAGES.
Shock
February 2025
Department of Respiratory and Critical Care Medicine, The First Affiliated Hospital of Wenzhou Medical University, Key Laboratory of Interventional Pulmonology of Zhejiang Province, Wenzhou, Zhejiang Province, China.
Objective: The mechanisms underlying the increased severity of hypertriglyceridemia acute pancreatitis (HTG-AP) remain poorly understood. Fibrinogen-like protein 2 (FGL2) has been identified as a regulator of macrophage activity, mediating immune suppression. This study aims to examine the role of FGL2 in the susceptibility to severe conditions of HTG-AP.
View Article and Find Full Text PDFHypertriglyceridemia Results From an Impaired Catabolism of Triglyceride-Rich Lipoproteins in -Related Lipodystrophy.
Arterioscler Thromb Vasc Biol
August 2024
Inserm U938, Saint-Antoine Research Centre, Institute of Cardiometabolism and Nutrition, Sorbonne University, Paris, France (E.C., C.V.).
Background: Pathogenic variants in -encoding PLIN1 (perilipin-1) are responsible for an autosomal dominant form of familial partial lipodystrophy (FPL) associated with severe insulin resistance, hepatic steatosis, and important hypertriglyceridemia. This study aims to decipher the mechanisms of hypertriglyceridemia associated with -related FPL.
Methods: We performed an in vivo lipoprotein kinetic study in 6 affected patients compared with 13 healthy controls and 8 patients with type 2 diabetes.
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