AI Article Synopsis
- - The study aimed to explore the relationship between dietary potassium intake and abdominal aortic calcification (AAC) and arterial stiffness in adults over 40, using data from the National Health and Nutrition Examination Survey (2013-2014).
- - Results showed that higher potassium intake wasn't linearly linked to AAC levels, but those in the second quartile had less severe AAC compared to the lowest quartile, along with significantly lower pulse pressure.
- - While no direct linear relationship was found for AAC, increased dietary potassium was correlated with lower pulse pressure, indicating potential benefits for arterial stiffness.
Article Abstract
Objectives: Arterial calcification contributes to cardiovascular mortality. Based on a recent animal study, we hypothesized that higher dietary potassium intake was associated with less abdominal aortic calcification (AAC) and lower arterial stiffness among adults in the United States.
Methods: Cross-sectional analyses were performed on participants over 40 years old from the National Health and Nutrition Examination Survey 2013-2014. Dietary potassium intake was categorized into quartiles (Q1: <1911, Q2: 1911-2461, Q3: 2462-3119, and Q4: >3119 mg/d). Primary outcome AAC was quantified using the Kauppila scoring system. AAC scores were categorized into no AAC (AAC = 0, reference group), mild/moderate (AAC >0 to ≤ 6), and severe AAC (AAC >6). Pulse pressure was used as a surrogate for arterial stiffness and examined as a secondary outcome.
Results: Among 2,418 participants, there was not a linear association between dietary potassium intake and AAC. Higher dietary potassium intake was associated with less severe AAC when comparing dietary potassium intake in Q2 with Q1 (odds ratio 0.55; 95% confidence interval: 0.34 to 0.92; P = .03). Higher dietary potassium intake was significantly associated with lower pulse pressure (P = .007): per 1000 mg/d higher dietary potassium intake, pulse pressure was 1.47 mmHg lower in the fully adjusted model. Compared to participants with dietary potassium intake in Q1, pulse pressure was 2.84 mmHg lower in Q4 (P = .04).
Conclusions: We did not find a linear association between dietary potassium intake and AAC. Dietary potassium intake was negatively associated with pulse pressure.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC10528025 | PMC |
| http://dx.doi.org/10.1053/j.jrn.2023.06.003 | DOI Listing |
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