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| http://dx.doi.org/10.3724/abbs.2023077 | DOI Listing |
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Demethoxycurcumin represses cervical cancer growth through PPARγ-regulated proliferation and apoptosis.
Acta Biochim Biophys Sin (Shanghai)
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Department of Anesthesiology, Hunan Provincial Maternal and Child Health Care Hospital, Changsha 410008, China.
Demethoxycurcumin induces apoptosis in HER2 overexpressing bladder cancer cells through degradation of HER2 and inhibiting the PI3K/Akt pathway.
Environ Toxicol
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Department of Biological Science and Technology, College of Life Sciences, China Medical University, Taichung, Taiwan.
Bladder cancer is the most common malignancy of the urinary tract and arising from the epithelial lining of the urinary bladder. Resistance to cytotoxic therapies is associated with overexpression of oncogenic proteins; including HER2, and Akt in chemotherapy resistance of bladder cancer. Various studies demonstrated that curcuminoids, the most important active phenolic compounds of turmeric (Curcuma longa), have anti-tumor activities in a wide range of human malignant cell lines.
View Article and Find Full Text PDFCurcumin, demethoxycurcumin, and bisdemethoxycurcumin induced caspase-dependent and -independent apoptosis via Smad or Akt signaling pathways in HOS cells.
BMC Complement Med Ther
March 2020
Department of Applied Science, National Tsing Hua University South Campus, No.521, Nanda Rd, Hsinchu City, 30014, Taiwan.
Background: Osteosarcoma is the most common primary malignant bone tumor in children and adolescents and has also been associated with a high degree of malignancy and enhanced metastatic capacity. Curcumin (CUR) is well known for its anti-osteosarcoma activity. However, both demethoxycurcumin (DMC), and bisdemethoxycurcumin (BDMC) are natural curcumin analogues/congeners from turmeric whose role in osteosarcoma development remains unknown.
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