The authors substantiate the assumption that the presence of high-amplitude superslow potential fluctuations in registration from the skin surface of the head is determined by destabilization of subcortical intercentral relationships which comprises inhibition of stem and activation of posterohypothalamic structures in craniocerebral trauma. The constant potential reacts only to the sharpest period of a severe craniocerebral trauma and reflects acute inflammatory and vascular phenomena on the cortical level in the first 24 hours; maintenance or appearance of increased positive values of the constant potential over one of the hemispheres suggests the presence of an intracranial hematoma above this hemisphere.
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