Activation of the external pathway (amytalantimycine A-resistant) of the exogenous NADH oxidation was studied in rat liver mitochondria after long-term (within 6 days) administration of pyracetame. Stimulation of lipid peroxidation and a decrease in viscosity of mitochondrial membranes accompanied the increase in the rate of oxygen consumption in mitochondria in presence of the exogenous NADH, amytal and antimycine A.
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