AI Article Synopsis
- Cerebral hyperperfusion syndrome (CHS) can occur as a complication following cerebral revascularization due to impaired cerebral autoregulation after long-term reduced blood flow.
- A case study of a 40-year-old man with symptomatic intracranial arterial dissection highlighted the risk of CHS after he underwent stenting for severe cerebral hypoperfusion.
- The findings suggest that even short-term cerebral hypoperfusion can trigger CHS, emphasizing the importance of monitoring patients closely post-procedure.
Article Abstract
Cerebral hyperperfusion syndrome (CHS) is one of the complications of cerebral revascularization. The main pathophysiology of CHS was considered to be cerebral autoregulation impairment due to long-standing cerebral hypoperfusion. Herein, we describe the case of a 40-year-old man with symptomatic intracranial arterial dissection (IAD) related to internal carotid artery stenosis. The patient underwent intracranial stenting 11 days after onset due to severe cerebral hypoperfusion presenting with neurological symptoms, and CHS presenting with intracerebral hemorrhage, post-operatively. The present case indicated not only the potential risk of CHS after intracranial stenting in IAD-related stenosis but also that cerebral hypoperfusion-even in a short period-might lead to CHS.
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| http://dx.doi.org/10.1016/j.clineuro.2023.107667 | DOI Listing |
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