Bhartiya S. Niacinamide and Neuroprotection: The Glaucoma Holy Grail. J Curr Glaucoma Pract 2022;16(3):141-143.
Download full-text PDF |
Source |
---|---|
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC9905873 | PMC |
http://dx.doi.org/10.5005/jp-journals-10078-1390 | DOI Listing |
Brain Behav
January 2025
Department of Emergency and Trauma Center, Nanchang First Hospital, Nanchang, Jiangxi, China.
Introduction: Depression is a prevalent and significant psychological consequence of traumatic brain injury (TBI). Ferroptosis, an iron-dependent form of regulated cell death, exacerbates the neurological damage associated with TBI. This study investigates whether nicorandil, a potassium channel opener with nitrate-like properties known for its antioxidative and neuroprotective effects, can mitigate depression-like behaviors following TBI by modulating ferroptosis.
View Article and Find Full Text PDFRapid Commun Mass Spectrom
February 2025
Department of Neurology, Affiliated Hospital of Inner Mongolia Medical University, Hohhot, China.
Neurochem Res
November 2024
I. Horbachevsky Ternopil National Medical University, Ministry of Health of Ukraine, Ternopil, Ukraine.
Diabetes is a multifunctional chronic disease that affects both the central and/or peripheral nervous systems. This study assessed whether nicotinamide (NAm) or conjugate of nicotinic acid with gamma-aminobutyric acid (N-GABA) could be potential neuroprotective agents against type 1 diabetes (T1D)-induced nervous system impairments in rats. After six weeks of T1D, induced by streptozotocin, nonlinear male Wistar rats were treated for two weeks with NAm (100 mg/kg, i.
View Article and Find Full Text PDFZh Nevrol Psikhiatr Im S S Korsakova
October 2024
Tashkent Pediatric Medical Institute, Tashkent, Uzbekistan.
Objective: Evaluation of the effectiveness of Cytoflavin as a neuroprotective therapy for neurological complications in premature newborns with hypoxic-ischemic encephalopathy.
Material And Methods: 56 premature newborns with low body weight were examined. Newborn children, depending on the therapy, were divided into 2 groups: Group I - 32 premature newborns with hypoxic-ischemic encephalopathy; received basic therapy and Cytoflavin; Group II - 24 premature newborns with hypoxic-ischemic encephalopathy; received only basic therapy.
Neurobiol Dis
November 2024
Department of Neurogenetics, Center for Development of Advanced Medicine for Dementia, National Center for Geriatrics and Gerontology, Obu, Aichi, Japan; Department of Experimental Gerontology, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan. Electronic address:
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!