Severity: Warning
Message: file_get_contents(https://...@pubfacts.com&api_key=b8daa3ad693db53b1410957c26c9a51b4908&a=1): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests
Filename: helpers/my_audit_helper.php
Line Number: 176
Backtrace:
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 176
Function: file_get_contents
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 250
Function: simplexml_load_file_from_url
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 1034
Function: getPubMedXML
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 3152
Function: GetPubMedArticleOutput_2016
File: /var/www/html/application/controllers/Detail.php
Line: 575
Function: pubMedSearch_Global
File: /var/www/html/application/controllers/Detail.php
Line: 489
Function: pubMedGetRelatedKeyword
File: /var/www/html/index.php
Line: 316
Function: require_once
As a direct carbon emission source, the amount of nitrous oxide (N, which is actually caused by AOB denitrification. To control the NO emission during biological N-removal, complete HND and NO accumulation for AOB denitrification should be avoided to a large extent. For this purpose, DO in aerobic tanks should be controlled at a normal level (approximately 2 mg·L), and solid retention time (SRT) should be extended, up to 20 d, which would avoid accumulating NO for AOB denitrification. Additionally, external carbon should be supplemented in time to promote HDN approaching the end, N. This review summarizes the mechanisms of all the mentioned NO emission pathways and discusses the control strategies of NO emission according to the associated mechanisms.
Download full-text PDF |
Source |
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http://dx.doi.org/10.13227/j.hjkx.202204041 | DOI Listing |
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