Current artificial intelligence systems for determining a person's emotions rely heavily on lip and mouth movement and other facial features such as eyebrows, eyes, and the forehead. Furthermore, low-light images are typically classified incorrectly because of the dark region around the eyes and eyebrows. In this work, we propose a facial emotion recognition method for masked facial images using low-light image enhancement and feature analysis of the upper features of the face with a convolutional neural network. The proposed approach employs the AffectNet image dataset, which includes eight types of facial expressions and 420,299 images. Initially, the facial input image's lower parts are covered behind a synthetic mask. Boundary and regional representation methods are used to indicate the head and upper features of the face. Secondly, we effectively adopt a facial landmark detection method-based feature extraction strategy using the partially covered masked face's features. Finally, the features, the coordinates of the landmarks that have been identified, and the histograms of the oriented gradients are then incorporated into the classification procedure using a convolutional neural network. An experimental evaluation shows that the proposed method surpasses others by achieving an accuracy of 69.3% on the AffectNet dataset.
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http://dx.doi.org/10.3390/s23031080 | DOI Listing |
Behav Res Methods
January 2025
Department of Psychology, University of Quebec at Trois-Rivières, Trois-Rivières, Canada.
Frequently, we perceive emotional information through multiple channels (e.g., face, voice, posture).
View Article and Find Full Text PDFAlzheimers Dement
December 2024
University of Wollongong, Wollongong, NSW, Australia.
Background: Brain iron dyshomeostasis has been observed in behavioral deficits relevant to neurodegenerative diseases such as Alzheimer's disease (AD), but it remains unclear whether it is a primary cause or an epiphenomenon of disease.
Method: We assessed the effects of brain iron dyshomeostasis on spatial cognition and cognitive flexibility using the IntelliCage system, recognition memory using novel object recognition tasks and anxiety-like behavior using the open field and elevated plus maze tests. We investigated these phenotypes in a HfexTfr2 mouse model of brain iron dyshomeostasis alone (Iron) or combined with an APP/PS1 model of Alzheimer's Aβ amyloidosis (Aβ+Iron), compared with APP/PS1 mice with Aβ amyloidosis alone (Aβ) or wildtype controls.
Alzheimers Dement
December 2024
University of Iowa, Iowa City, IA, USA.
Background: Sorbs2 is a cytoskeletal adaptor protein that is expressed in hippocampal neurons, but its mechanistic role in these cells is not yet fully understood.
Method: We created two groups of mice for our study: whole-body Sorbs2-Knockout (KO) mice and Sorbs2-Flox mice, which had neuronal knockout via AAV-PHP.eB-hSyn1-Cre virus injection.
Alzheimers Dement
December 2024
Laboratory of Cellular Neurobiology, Departamento de Fisiologia e Biofísica, Instituto de Ciências Biomédicas, Universidade de São Paulo, São Paulo, São Paulo, Brazil.
Background: In Alzheimer's disease (AD) the fact that neuropsychiatric symptoms can predate the onset of cognitive symptoms suggests that greater focus on the non-cognitive behavioral changes in earlier life could be an opportunity to investigate 'latent' mild behavioral impairment (MBI) as a possible diagnostic strategy for preclinical AD.
Method: We used 1- and 6-month-old 3xTg-AD male mice and age-matched wild-type animals (CEUA-ICB/USP: 127/2015). Two batteries of behavioral tests were performed: (1) open field test (OFT), novel object recognition test (NORT), and rotarod test; (2) elevated zero maze test (EZMT), forced swim test (FST), and sucrose preference test (SPT).
Alzheimers Dement
December 2024
Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Background: The salience network (SN) functions as a dynamic switch between the default mode network (DMN) and the frontoparietal network (FPN), aligning with salience and cognitive demand. Dysfunctions in SN activity within the cognitive and affective domains are linked to a wide range of deficits and maladaptive behavioral patterns in various clinical disorders. Emotion recognition is pivotal in social interactions and can be affected in neurodegenerative disorders.
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