To ensure that computers can accomplish specific tasks intelligently and autonomously, it is common to introduce more knowledge into artificial intelligence (AI) technology as prior information, by imitating the structure and mindset of the human brain. Currently, unmanned aerial vehicle (UAV) tracking plays an important role in military and civilian fields. However, robust and accurate UAV tracking remains a demanding task, due to limited computing capability, unanticipated object appearance variations, and a volatile environment. In this paper, inspired by the memory mechanism and cognitive process in the human brain, and considering the computing resources of the platform, a novel tracking method based on Discriminative Correlation Filter (DCF) based trackers and memory model is proposed, by introducing dynamic feature-channel weight and aberrance repressed regularization into the loss function, and by adding an additional historical model retrieval module. Specifically, the feature-channel weight integrated into the spatial regularization (SR) enables the filter to select features. The aberrance repressed regularization provides potential interference information to the tracker and is advantageous in suppressing the aberrances caused by both background clutter and appearance changes of the target. By optimizing the aforementioned two jointly, the proposed tracker could restrain the potential distractors, and train a robust filter simultaneously by focusing on more reliable features. Furthermore, the overall loss function could be optimized with the Alternative Direction Method of Multipliers (ADMM) method, thereby improving the calculation efficiency of the algorithm. Meanwhile, with the historical model retrieval module, the tracker is encouraged to adopt some historical models of past video frames to update the tracker, and it is also incentivized to make full use of the historical information to construct a more reliable target appearance representation. By evaluating the method on two challenging UAV benchmarks, the results prove that this tracker shows superior performance compared with most other advanced tracking algorithms.
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http://dx.doi.org/10.3389/fnins.2022.1080521 | DOI Listing |
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January 2025
Department of Biochemistry, Faculty of Pharmacy, Badr University in Cairo (BUC), Badr City, , 11829, Cairo, Egypt.
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View Article and Find Full Text PDFTheriogenology
January 2025
College of Agriculture and Bioengineering, Heze University, Heze, 274000, China. Electronic address:
Precisely regulated spermatocyte growth, differentiation, and apoptosis are crucial for sustainable male fertility. miR-143 has been demonstrated to regulate gene expression and cell apoptosis in various human cancers. However, the function of mmu-mir-143 (miR-143) in mammalian testes and its underlying mechanism remains unexplored.
View Article and Find Full Text PDFCell Rep
January 2025
Department of Basic Medical Sciences, School of Medicine, Xiamen University, Xiamen, Fujian, P.R. China; State Key Laboratory of Cellular Stress Biology, Xiamen University, Xiamen, Fujian, P.R. China. Electronic address:
Menin is a scaffold protein encoded by the Men1 gene, and it interacts with a variety of chromatin regulators to activate or repress cellular processes. The potential importance of menin in immune regulation remains unclear. Here, we report that myeloid deletion of Men1 results in the development of spontaneous pulmonary alveolar proteinosis (PAP).
View Article and Find Full Text PDFAging Cell
January 2025
Temasek Life Sciences Laboratory, Singapore, Singapore.
Multimodal study of Alzheimer's disease (AD) dorsolateral prefrontal cortex (DLPFC) showed AD-related aberrant intron retention (IR) and proteomic changes not observed at the RNA level. However, the role of sex and how IR may impact the proteome are unclear. Analysis of DLPFC transcriptome showed a clear sex-biased pattern where female AD had 1645 elevated IR events compared to 80 in male AD DLPFC.
View Article and Find Full Text PDFWhile inputs regulating CD4 T helper cell (Th) differentiation are well-defined, the integration of downstream signaling with transcriptional and epigenetic programs that define Th-lineage identity remain unresolved. PI3K signaling is a critical regulator of T cell function; activating mutations affecting PI3Kδ result in an immunodeficiency with multiple T cell defects. Using mice expressing activated-PI3Kδ, we found aberrant expression of proinflammatory Th1-signature genes under Th2-inducing conditions, both and .
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