We present an example of bradycardia-dependent aberrancy in the left posterior fascicle in the presence of type I second-degree atrioventricular block occurring in the setting of acute inferior myocardial infarction. The need for prophylactic pacing of such cases is stressed.
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Aberrant Ventricular Conduction: Revisiting an Old Concept.
Heart Lung Circ
May 2023
CardioScan Pty Ltd, 293 Camberwell Rd, Melbourne, Vic, Australia; Department of Cardiology, The Royal Melbourne Hospital and the Department of Medicine, University of Melbourne, Vic, Australia. Electronic address:
Article Synopsis
- Aberrant ventricular conduction, a concept introduced over 100 years ago, is often misunderstood despite advancements in cardiac science, as it primarily originates from the His-Purkinje system's physiological refractoriness rather than true conduction system disease.
- This phenomenon can present itself through various patterns such as premature atrial ectopics and the Ashman phenomenon, which complicate the diagnosis and management of broad complex tachycardia.
- Recognizing and differentiating aberrant ventricular conduction from ventricular tachyarrhythmias is crucial, as they have different treatment strategies and prognostic outcomes; the review aims to clarify ECG characteristics for precise interpretation.
[Intermittent bundle branch block: a clinical model for the study of electrophysiological phenomena].
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January 2014
Disorders of intraventricular conduction (bundle branch block and hemiblock) are usually stable and remain unchanged irrespective of heart rate. Not infrequently, however, their appearance is related to the duration of the cardiac cycle, so that they appear and disappear with changes in heart rate. This may not even represent a pathological phenomenon, since sudden and consistent changes in cardiac cycle can result, even physiologically, in aberrant conduction.
View Article and Find Full Text PDFTen years of genes in inherited arrhythmia syndromes: an example of what we have learned from patients, electrocardiograms, and computers.
J Electrocardiol
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Department of Clinical and Experimental Cardiology, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands.
In the last 10 years the molecular substrate of a diversity of primary electrical diseases has been unraveled. Disease-causing mutations in ion-channel genes have been identified and have challenged clinical and basic electrophysiologists to mechanistically link the observed genetic aberrancies to the phenotype. Indeed, in collaborative efforts of clinical cardiologists, clinical and molecular geneticists, basic electrophysiologists and computer modellers, the pathophysiology of many clinical electrocardiographic findings have been elucidated in detail.
View Article and Find Full Text PDFAberrant ventricular conduction is a rare phenomenon as compared with the more frequently occurring antrioventricular conduction disturbances. It leads to widening of the QRS complex, which is either due to a complete or functional block in one of the bundle branches or a block within the intramyocardial conduction system itself. Mechanisms that are potentially involved in the genesis of aberrant ventricular conduction are sudden shortening of cycle length (tachycardia-dependent phase III), antegrade block with retrograde concealed conduction, or bradycardia-dependent block (enhanced phase IV).
View Article and Find Full Text PDF[A-V conduction in atrial fibrillation and flutter].
Cardiologia
August 1991
Cattedra di Malattie Cardiovascolari, Università degli Studi, Messina.
The assessment of A-V conduction in the presence of atrial fibrillation is based upon analysis of the R-R intervals. This is because in atrial fibrillation it is impossible both to identify the impulse that has been conducted to the ventricles, and to measure the A-V conduction time. The first step is, therefore, to evaluate whether the QRS complexes are the expression of conducted atrial impulses, or they are A-V junctional or ventricular in origin.
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