Objectives: While self-compassion (SC) has mostly been understood as a stable trait-like property, growing evidence suggests that it may fluctuate over time within a given individual. However, little is known on how these fluctuations relate to affective well-being and affective dynamics, such as emotional inertia and stress reactivity in daily life.

Methods: A sample of 119 non-clinical individuals (mean age: 31.3 years, 53.8% female) completed a 7-day smartphone-based ecological momentary assessment study with six semi-random signals per day. With each signal, individuals reported their momentary positive affect (PA) and negative affect (NA), recent SC, and occurrence and perceived strain of daily hassles since the last signal.

Results: Whenever individuals reported higher recent SC than usual, they experienced higher momentary PA and lower momentary NA. Moreover, higher recent SC related to lower stress reactivity in terms of lower decrease of PA and lower increase of NA following the experience of daily hassles. No associations between SC and emotional inertia were found. When distinguishing between the positive components (SC-Pos) and negative components (SC-Neg) of SC, SC-Neg (compared to SC-Pos) was more strongly connected to NA, while SC-Pos and SC-Neg were similarly connected to PA. SC-Pos was associated with an attenuated NA stress reactivity, and SC-Neg with an increased NA stress reactivity. SC-Pos and SC-Neg did not significantly moderate PA stress reactivity nor emotional inertia.

Conclusions: Results show that the benefits of SC for well-being and stress reactivity may unfold whenever we treat ourselves with compassion, irrespective of how self-compassionate we are in general.

Supplementary Information: The online version contains supplementary material available at 10.1007/s12671-022-02050-y.

Download full-text PDF

Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC9823261PMC
http://dx.doi.org/10.1007/s12671-022-02050-yDOI Listing

Publication Analysis

Top Keywords

stress reactivity
24
affective dynamics
8
emotional inertia
8
individuals reported
8
daily hassles
8
connected sc-pos
8
sc-pos sc-neg
8
stress
6
reactivity
6
momentary
5

Similar Publications

Background: Postmenopausal women (PMW) who complete menopause at a late age (55+ years) have lower cardiovascular disease risk than PMW who complete menopause at a normal age (45-54 years). However, the influence of late-onset menopause on vascular endothelial dysfunction is unknown. Moreover, the mechanisms by which a later age at menopause may modulate endothelial function remain to be determined.

View Article and Find Full Text PDF

L. fruits and leaf extracts have a broad range of immunomodulatory, anti-inflammatory, and antioxidant effects; however, their effects on cardiac protection have not been investigated. The study aims to test the biological activity of L.

View Article and Find Full Text PDF

CO-loaded hemoglobin/EGCG nanoparticles functional coatings for inflammation modulation of vascular implants.

Regen Biomater

December 2024

Institute of Biomedical Engineering, College of Medicine, Southwest Jiaotong University, Chengdu, Sichuan 610031, China.

During the implantation process of cardiovascular implants, vascular damage caused by inflammation occurs, and the inflammatory process is accompanied by oxidative stress. Currently, carbon monoxide (CO) has been demonstrated to exhibit various biological effects including vasodilatation, antithrombotic, anti-inflammatory, apoptosis-inducing and antiproliferative properties. In this study, hemoglobin/epigallocatechin-3-gallate (EGCG) core-shell nanoparticle-containing coating on stainless steel was prepared for CO loading and inflammation modulation.

View Article and Find Full Text PDF

Oxidative stress contributes to the development of cardiometabolic diseases and cancers. Numerous studies have highlighted the adverse effects of high reactive oxygen species (ROS) levels in the progression of chronic noncommunicable diseases and also during infections. On the other hand, antioxidants play a crucial role in preventing oxidative stress or postponing cell damage via the direct scavenging of free radicals or indirectly via the Keap1/Nrf2/ARE pathway, among others.

View Article and Find Full Text PDF

Background: A primary factor in the pathogenesis of aging is oxidative stress, with cardiac inflammation and fibrosis being contributed to by increased oxidative stress as organisms age. Oxidative stress enhances the cardiac fibrotic signaling pathway, with reactive oxygen species inducing cardiac fibrosis through increased expression of the profibrotic factor transforming growth factor-beta 1 (TGF-β1). Furthermore, Wnt/β-catenin signaling pathway is implicated in interstitial fibrosis, which is associated with TGF-β.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!