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| http://dx.doi.org/10.1016/j.bbalip.2022.159260 | DOI Listing |
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increases Prostaglandin E secretion in cow neutrophils by activating TLR2, TLR4, and NLRP3 inflammasome signaling pathways.
Front Microbiol
April 2023
College of Veterinary Medicine, Inner Mongolia Agricultural University, Huhhot, China.
Introduction: In clinical settings, dairy cows are often attacked by pathogenic bacteria after delivery, especially (). Neutrophils have long been regarded as essential for host defense against . Prostaglandin E (PGE) can additionally be used as an inflammatory mediator in pathological conditions to promote the repair of inflammatory injuries.
View Article and Find Full Text PDFCommercial lipoprotein-deficient sera differ in iron content.
Biochim Biophys Acta Mol Cell Biol Lipids
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Department of Cell Biology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, United States of America. Electronic address:
Secretes a Bioactive Lipid That Triggers Inflammatory Signaling and Cell Death.
Front Microbiol
May 2022
Department of Infectious Diseases, Genentech, South San Francisco, CA, United States.
is a highly pathogenic Gram-negative bacterium that causes severe infections with very high fatality rates. infection triggers innate as well as adaptive immunity, however, our understanding of the inflammatory factors secreted by that alarm the immune system remains limited. In this study, we report that the lab adapted and clinical strains of secrete an inflammatory bioactive factor which activates TLR2, leading to canonical IRAK4-dependent NF-κB signaling and production of pro-inflammatory cytokines interleukin (IL)-6 and IL-8 and activation of the inflammasome pathway causing pyroptotic cell death.
View Article and Find Full Text PDFInduction of Apoptotic Cell Death by Oral Streptococci in Human Periodontal Ligament Cells.
Front Microbiol
October 2021
Department of Oral Microbiology and Immunology and Dental Research Institute, School of Dentistry, Seoul National University, Seoul, South Korea.
Initiation and progression of oral infectious diseases are associated with streptococcal species. Bacterial infection induces inflammatory responses together with reactive oxygen species (ROS), often causing cell death and tissue damage in the host. In the present study, we investigated the effects of oral streptococci on cytotoxicity and ROS production in human periodontal ligament (PDL) cells.
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