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                            [content] => *! The study examines the role of the p110β subunit of phosphatidylinositol 3-kinase in platelet-mediated immune responses during infections, particularly in the context of cancer treatments using p110β inhibitors. *! Using a mouse model of pneumonia caused by Streptococcus pneumoniae, researchers found that a deficiency or inhibition of platelet p110β worsened the disease by disrupting the interactions between platelets and immune cells like monocytes and neutrophils. *! The results indicate that p110β is crucial for effective immune responses during bacterial infections, suggesting that inhibiting p110β could negatively impact immune functions while undergoing anti-cancer therapies.
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