Particleboard surface defects have a significant impact on product quality. A surface defect detection method is essential to enhancing the quality of particleboard because the conventional defect detection method has low accuracy and efficiency. This paper proposes a YOLO v5-Seg-Lab-4 (You Only Look Once v5 Segmentation-Lab-4) model based on deep learning. The model integrates object detection and semantic segmentation, which ensures real-time performance and improves the detection accuracy of the model. Firstly, YOLO v5s is used as the object detection network, and it is added into the SELayer module to improve the adaptability of the model to receptive field. Then, the Seg-Lab v3+ model is designed on the basis of DeepLab v3+. In this model, the object detection network is utilized as the backbone network of feature extraction, and the expansion rate of atrus convolution is reduced to the computational complexity of the model. The channel attention mechanism is added onto the feature fusion module, for the purpose of enhancing the feature characterization capabilities of the network algorithm as well as realizing the rapid and accurate detection of lightweight networks and small objects. Experimental results indicate that the proposed YOLO v5-Seg-Lab-4 model has mAP (Mean Average Precision) and mIoU (Mean Intersection over Union) of 93.20% and 76.63%, with a recognition efficiency of 56.02 fps. Finally, a case study of the Huizhou particleboard factory inspection is carried out to demonstrate the tiny detection accuracy and real-time performance of this proposed method, and the missed detection rate of surface defects of particleboard is less than 1.8%.
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http://dx.doi.org/10.3390/s22207733 | DOI Listing |
Genet Med
December 2024
Sheffield Clinical Genetics Service, Sheffield Children's NHS Foundation Trust, Sheffield, UK; Division of Clinical Medicine, University of Sheffield, Sheffield, UK. Electronic address:
Purpose: The TAOK proteins are a group of serine/threonine-protein kinases involved in signalling pathways, cytoskeleton regulation, and neuronal development. TAOK1 variants are associated with a neurodevelopmental disorder (NDD) characterized by distinctive facial features, hypotonia and feeding difficulties. TAOK2 variants have been reported to be associated with autism and early-onset obesity.
View Article and Find Full Text PDFCureus
November 2024
Anesthesiology and Pain Medicine, Harborview Medical Center, Seattle, USA.
Prompt emergence from general anesthesia is crucial after neurosurgical procedures, such as craniotomies, to facilitate timely neurological evaluation for identification of intraoperative complications. Delayed emergence can be caused by residual anesthetics, metabolic imbalances, and intracranial pathology, for which an eye examination can provide early diagnostic clues. The sunset sign (or setting sun sign), characterized by a downward deviation of the eyes, can be an early indicator of raised intracranial pressure (ICP) or midbrain compression, as is commonly observed in states of hydrocephalus or periaqueductal or tectal plate dysfunction.
View Article and Find Full Text PDFArch Razi Inst
June 2024
Department of Physiology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.
Gastrointestinal dysfunction is a severe and common complication in diabetic patients. Some evidence shows that gamma-aminobutyric acid (GABA) and glutamate contribute to diabetic gastrointestinal abnormalities. Therefore, we examined the impact of prolonged treatment with insulin and magnesium supplements on the expression pattern of GABA type A (GABA-A), GABA-B, and N-methyl-D-aspartate (NMDA) glutamate receptors as well as nitric oxide synthase 1 (NOS-1) in the stomach of type 2 diabetic rats.
View Article and Find Full Text PDFFront Endocrinol (Lausanne)
December 2024
Rare Disease Research Group, Molecular (Epi) Genetics Laboratory, Bioaraba Health Research Institute, Araba University Hospital, Vitoria-Gasteiz, Spain.
Objective: To identify the genetic cause underlying the methylation defect in a patient with clinical suspicion of PHP1B/iPPSD3.
Design: Imprinting is an epigenetic mechanism that allows the regulation of gene expression. The locus is one of the loci within the genome that is imprinted.
J Integr Neurosci
December 2024
Department of Human Anatomy, School of Basic Medical Sciences, Wannan Medical College, 241002 Wuhu, Anhui, China.
Background: K48-linked ubiquitin chain (Ub-K48) is a crucial ubiquitin chain implicated in protein degradation within the ubiquitin-proteasome system. However, the precise function and molecular mechanism underlying the role of Ub-K48 in the pathogenesis of Alzheimer's disease (AD) and neuronal cell abnormalities remain unclear. The objective of this study was to examine the function of K48 ubiquitination in the etiology of AD, and its associated mechanism of neuronal apoptosis.
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