How genetic and environmental risk factors interact to trigger the development of post-traumatic stress disorder (PTSD) remains largely unknown. Seah et al. model stress hypersensitivity as a potential mechanism by examining transcriptomic responses to glucocorticoids in neurons derived from individuals with PTSD.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC9721095 | PMC |
| http://dx.doi.org/10.1038/s41593-022-01172-9 | DOI Listing |
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