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| http://dx.doi.org/10.1016/j.amsu.2022.104579 | DOI Listing |
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Background: glucocorticoids may play an important role in the formation of fear memory, which is relevant to the neurobiology of post-traumatic stress disorder (PTSD). In our previous study, we showed the glucocorticoid receptor (GR) forms a protein complex with FKBP51, which prevents translocation of GR into the nucleus to affect gene expression; this complex is elevated in PTSD patients and by fear-conditioned learning in mice, and disrupting this complex blocks the storage and retrieval of fear-conditioned memories. The timing of release of glucocorticoid relative to the formation of a traumatic memory could be important in this process, and remains poorly understood.
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Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus, Denmark.
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Nursing School, The University of Jordan, Amman, Jordan.
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