[This corrects the article DOI: 10.1039/D2SC00385F.].
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http://dx.doi.org/10.1039/d2sc90140d | DOI Listing |
Chem Sci
July 2022
TU Dortmund University, Department of Chemistry and Chemical Biology Otto-Hahn-Straße 6 44227 Dortmund Germany
[This corrects the article DOI: 10.1039/D2SC00385F.].
View Article and Find Full Text PDFTransl Pediatr
April 2019
Pediatric Heart Center of Hessen, Justus Liebig University, Giessen, Germany.
Critical congenital heart disease (cCHD) is the most common reason for acute cardiac failure in the neonatal period. cCHD, defined by systemic low cardiac output (LCO) and requiring surgery or catheter-based intervention in the first year of life, has an incidence of approximately 15% of CHD and is responsible for up to 25% fatalities of newborn infants. Clinical deterioration develops in most cases due to rapid closure of the ductus arteriosus (DA).
View Article and Find Full Text PDFCancer Res
August 1988
Institute of Human Genetics, Free University, Amsterdam, The Netherlands.
Based on the concept that activated oxygen species are causally involved in Adriamycin toxicity, endogenous antioxidant defenses are expected to be important determinants of cellular Adriamycin tolerance. We have tested this prediction by making use of an oxygen-resistant variant subline of Chinese hamster ovary cells (CHOr), which is characterized by increased levels of glutathione, copper- and zinc-containing superoxide dismutase, manganese-containing superoxide dismutase, catalase, and glutathione peroxidase. The levels of antioxidant defenses in wild-type CHO (CHOs) cells were within the range reported for human tumor cell lines, except for catalase, which was comparatively high.
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