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Verticillium dahliae Elicitor VdSP8 Enhances Disease Resistance Through Increasing Lignin Biosynthesis in Cotton.
Plant Cell Environ
January 2025
State Key Laboratory of Crop Stress Resistance and High-Efficiency Production, Key Laboratory of Plant Protection Resources and Pest Integrated Management of Ministry of Education, Key Laboratory of Integrated Pest Management on Crops in Northwestern Loess Plateau of Ministry of Agriculture and Rural Affairs, and College of Plant Protection, Northwest A&F University, Yangling, China.
Verticillium wilt caused by the soil-borne fungus Verticillium dahliae Kleb., is a destructive plant disease that instigates severe losses in many crops. Improving plant resistance to Verticillium wilt has been a challenge in most crops.
View Article and Find Full Text PDFHyper-Sensitive? Targeted Therapy With a Primed Immune System.
J Thorac Oncol
June 2022
Department of Medical Oncology, BC Cancer, Vancouver, British Columbia, Canada; Department of Medicine, University of British Columbia, Vancouver, British Columbia, Canada. Electronic address:
A Paradigm for Post-Covid-19 Fatigue Syndrome Analogous to ME/CFS.
Front Neurol
August 2021
The Brain Health Research Centre, University of Otago, Dunedin, New Zealand.
A significant proportion of COVID-19 patients are suffering from prolonged Post-COVID-19 Fatigue Syndrome, with characteristics typically found in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS). However, no clear pathophysiological explanation, as yet, has been provided. A novel paradigm for a Post-COVID-19 Fatigue Syndrome is developed here from a recent unifying model for ME/CFS.
View Article and Find Full Text PDFCurcumin arrests G-quadruplex in the nuclear hyper-sensitive III element of oncogene leading to apoptosis in metastatic breast cancer cells.
J Biomol Struct Dyn
December 2022
Department of Biophysics, Bose Institute, Kolkata, India.
is deregulated in triple negative breast cancer (TNBC) pointing to be a promising biomarker for breast cancer treatment. Precise level of expression is important in the control of cellular growth and proliferation. Designing of -targeted antidotes to restore its basal level of cellular expression holds an optimistic approach towards anti-cancer treatment.
View Article and Find Full Text PDFA noncanonical AR addiction drives enzalutamide resistance in prostate cancer.
Nat Commun
March 2021
Department of Biochemistry and Molecular Biology, Mayo Clinic College of Medicine and Science, Rochester, MN, USA.
Resistance to next-generation anti-androgen enzalutamide (ENZ) constitutes a major challenge for the treatment of castration-resistant prostate cancer (CRPC). By performing genome-wide ChIP-seq profiling in ENZ-resistant CRPC cells we identify a set of androgen receptor (AR) binding sites with increased AR binding intensity (ARBS-gained). While ARBS-gained loci lack the canonical androgen response elements (ARE) and pioneer factor FOXA1 binding motifs, they are highly enriched with CpG islands and the binding sites of unmethylated CpG dinucleotide-binding protein CXXC5 and the partner TET2.
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